(Q47990590)

7 February 2018

title

Inhibition of endothelial nitric oxide synthase induces and enhances myocardial fibrosis. (English)

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7 February 2018

author name string

Andrey Kazakov

1

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7 February 2018

Rabea Hall

2

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7 February 2018

Philippe Jagoda

3

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7 February 2018

Katrin Bachelier

4

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7 February 2018

Patrick Müller-Best

5

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7 February 2018

Alexander Semenov

6

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7 February 2018

Frank Lammert

7

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7 February 2018

Michael Böhm

8

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7 February 2018

Ulrich Laufs

9

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7 February 2018

publication date

17 July 2013

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7 February 2018

published in

Cardiovascular Research

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7 February 2018

volume

100

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7 February 2018

issue

2

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7 February 2018

page(s)

211-221

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7 February 2018

Nitric oxide synthases: regulation and function

cites work

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Nitric oxide and nitric oxide synthase isoforms in the normal, hypertrophic, and failing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Oxidant stress from nitric oxide synthase-3 uncoupling stimulates cardiac pathologic remodeling from chronic pressure load

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Reversal of cardiac hypertrophy and fibrosis from pressure overload by tetrahydrobiopterin: efficacy of recoupling nitric oxide synthase as a therapeutic strategy

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Pressure overload-induced LV hypertrophy and dysfunction in mice are exacerbated by congenital NOS3 deficiency.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Concentric left ventricular remodeling in endothelial nitric oxide synthase knockout mice by chronic pressure overload

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Endothelial nitric oxide synthase of the bone marrow regulates myocardial hypertrophy, fibrosis, and angiogenesis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Regulation of cardiac fibroblast extracellular matrix production by bradykinin and nitric oxide

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Nicorandil inhibits angiotensin-II-induced proliferation of cultured rat cardiac fibroblasts.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Angiotensin-(1-7) suppresses the number and function of the circulating fibrocytes by upregulating endothelial nitric oxide synthase expression

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

The origin of fibroblasts and mechanism of cardiac fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Bone marrow-derived fibroblast precursors mediate ischemic cardiomyopathy in mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Endothelial-to-mesenchymal transition contributes to cardiac fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Bone marrow-derived cells contribute to fibrosis in the chronically failing heart

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Myocardial fibrosis in response to Angiotensin II is preceded by the recruitment of mesenchymal progenitor cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Circulating fibrocytes traffic to the lungs in response to CXCL12 and mediate fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Fibrocyte CXCR4 regulation as a therapeutic target in pulmonary fibrosis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

CXCR4 antagonism attenuates the cardiorenal consequences of mineralocorticoid excess

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Selective pharmacological inhibition of distinct nitric oxide synthase isoforms

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

1400W is a slow, tight binding, and highly selective inhibitor of inducible nitric-oxide synthase in vitro and in vivo

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Antagonism of stromal cell-derived factor-1alpha reduces infarct size and improves ventricular function after myocardial infarction

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Inducible nitric oxide synthase deficiency protects the heart from systolic overload-induced ventricular hypertrophy and congestive heart failure.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

Pressure-induced cardiac overload induces upregulation of endothelial and myocardial progenitor cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FCVR%2FCVT181

21 January 2018

Identifiers

DOI

10.1093/CVR/CVT181

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7 February 2018

PubMed ID

23863197

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7 February 2018