(Q48036189)

English

Does the mechanism of protection from falciparum malaria by red cell genetic disorders involve a switch to a balanced TH1/TH2 cytokine production mode?

scientific article published in January 1997

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scholarly article

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Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

Does the mechanism of protection from falciparum malaria by red cell genetic disorders involve a switch to a balanced TH1/TH2 cytokine production mode? (English)

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

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Plasmodium falciparum

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1

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

publication date

1 January 1997

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

Medical Hypotheses

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

48

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

1

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

11-17

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

Protection afforded by sickle-cell trait against subtertian malareal infection

1 reference

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Malaria and human polymorphisms

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Increased sickling of parasitised erythrocytes as mechanism of resistance against malaria in the sickle-cell trait

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Cellular mechanism for the protective effect of haemoglobin S against P. falciparum malaria

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Oxidant damage mediates variant red cell resistance to malaria

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Impaired parasite growth and increased susceptibility to phagocytosis of Plasmodium falciparum infected alpha-thalassemia or hemoglobin Constant Spring red blood cells

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Unrestricted growth of Plasmodium falciparum in microcytic erythrocytes in iron deficiency and thalassaemia

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The sickle-cell trait modifies the intensity and specificity of the immune response against P. falciparum malaria and leads to acquired protective immunity

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Antibodies to blood stage antigens of Plasmodium falciparum in rural Gambians and their relation to protection against infection

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Surface antigen expression on Plasmodium falciparum-infected erythrocytes is modified in alpha- and beta-thalassemia

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Protection by alpha-thalassaemia against Plasmodium falciparum malaria: modified surface antigen expression rather than impaired growth or cytoadherence

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Erythrophagocytosis in malaria: Host defence or menace to the macrophage?

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The involvement of hemozoin toxicity in depression of cellular immunity.

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Cell-mediated immune responses to Plasmodium falciparum purified soluble antigens in sickle-cell trait subjects

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Lymphoproliferative Responses to Plasmodium falciparum Antigens in Children With and Without the Sickle Cell Trait

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Modulation of the cellular immune response during Plasmodium falciparum infections in sickle cell trait individuals

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Transgenic and mutant animal models to study mechanisms of protection of red cell genetic defects against malaria

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Pathology of membrane proteins in sickle erythrocytes

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Naturally occurring anti-band 3 autoantibodies recognize a high molecular weight protein on the surface of Plasmodium falciparum infected erythrocytes

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Antigens on the Plasmodium falciparum infected erythrocyte surface are parasite derived: a reply

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Antigens induced on erythrocytes by P. falciparum: expression of diverse and conserved determinants

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Protection against malaria by thalassaemia and haemoglobin variants.

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Impairment of macrophage functions after ingestion of Plasmodium falciparum-infected erythrocytes or isolated malarial pigment.

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The spleen in malaria: the role of barrier cells

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Reduced cellular immune reactivity in healthy individuals during the malaria transmission season

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Morbidity from malaria and immune responses to defined Plasmodium falciparum antigens in children with sickle cell trait in The Gambia

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Loss of cellular immune reactivity during acutePlasmodium falciparummalaria

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Transient depletion of T cells with high LFA-1 expression from peripheral circulation during acute Plasmodium falciparum malaria.

1 reference

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Human TH1 and TH2 subsets: doubt no more

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Centrefold: T-cell subsets and cytokines in parasitic infections

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Cytokines: making the right choice

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Induction of TH1 and TH2 responses: a key role for the 'natural' immune response?

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The role of CD4+ T-cells in the immune response to Plasmodium chabaudi

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Cell-mediated immunity to the asexual blood stages of malarial parasites: animal models

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Protective CD4+ T-cell lines raised against Plasmodium chabaudi show characteristics of either Th1 or Th2 cells

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Differential induction of helper T cell subsets during blood-stage Plasmodium chabaudi AS infection in resistant and susceptible mice

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Helper T-cell subsets in mouse trichuriasis

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Evasion strategies of Leishmania parasites.

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Establishment of stable, cell-mediated immunity that makes "susceptible" mice resistant to Leishmania major.

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T cell responses in acute falciparum malaria

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Malaria exoantigens induce TNF, are toxic and are blocked by T-independent antibody

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Significance of cytokine production and adhesion molecules in malarial immunopathology

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Role of cytokines and adhesion molecules in malaria immunopathology

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A TH1-->TH2 switch is a critical step in the etiology of HIV infection

1 reference

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Plasmodium falciparum malaria and human red cells. II. Red cell genetic traits and resistance against malaria

1 reference

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Effect of Sickle-cell Trait on Resistance to Malaria

1 reference

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Malaria and sickle-cell disease

1 reference

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Functional asplenia in sickle-cell anemia

1 reference

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Pathology of malaria in West Africa.

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Identifiers

10.1016/S0306-9877(97)90017-7

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

7 February 2018

http://europepmc.org/abstract/MED/9049983

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