(Q48087961)

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Recovery after nigral grafting in 6-hydroxydopamine lesioned rats is due to graft function and not significantly influenced by the remaining ipsilateral or contralateral host dopaminergic system.

scientific article published in September 1999

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1 November 2019

Recovery after nigral grafting in 6-hydroxydopamine lesioned rats is due to graft function and not significantly influenced by the remaining ipsilateral or contralateral host dopaminergic system (English)

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1 November 2019

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Elena Lopez-Martin

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1 November 2019

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G Rozas

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1 November 2019

M J Guerra

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1 November 2019

J L Labandeira-Garcia

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1 September 1999

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1 November 2019

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1 November 2019

842

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1 November 2019

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1 November 2019

119-131

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1 November 2019

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Neuropeptide messenger RNA expression in the 6-hydroxydopamine-lesioned rat striatum reinnervated by fetal dopaminergic transplants: differential effects of the grafts on preproenkephalin, preprotachykinin and prodynorphin messenger RNA levels

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Transection of corticostriatal afferents abolishes the hyperexpression of Fos and counteracts the development of rotational overcompensation induced by intrastriatal dopamine-rich grafts when challenged with amphetamine

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7 January 2021

Dopaminergic transplants normalize amphetamine- and apomorphine-induced Fos expression in the 6-hydroxydopamine-lesioned striatum

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Evidence for crossed catecholaminergic nigrostriatal projections by combining wheat germ agglutinin-horseradish peroxidase retrograde transport and tyrosine hydroxylase immunocytochemistry

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Behavioural recovery following transplantation of substantia nigra in rats subjected to 6-OHDA lesions of the nigrostriatal pathway. I. Unilateral lesions.

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7 January 2021

Behavioural recovery following transplantation of substantia nigra in rats subjected to 6-OHDA lesions of the nigrostriatal pathway. II. Bilateral lesions

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Dopamine-rich grafts ameliorate whole body motor asymmetry and sensory neglect but not independent limb use in rats with 6-hydroxydopamine lesions.

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Evidence for a crossed nigrostriatal pathway in rats

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Adrenal medullary autografts into the basal ganglia of Cebus monkeys: injury-induced regeneration.

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Functional recovery in parkinsonian monkeys treated with GDNF

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The neostriatal mosaic: multiple levels of compartmental organization

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Amphetamine and cocaine induce drug-specific activation of the c-fos gene in striosome-matrix compartments and limbic subdivisions of the striatum

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Previous dopaminergic innervation is not necessary for the development of dopamine supersensitivity in rat striatal neurons.

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Interaction between the serotonergic, dopaminergic, and glutamatergic systems in fenfluramine-induced Fos expression in striatal neurons

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Does adrenal graft enhance recovery of dopaminergic neurons in Parkinson's disease?

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A 16-channel automated rotometer system for reliable measurement of turning behavior in 6-hydroxydopamine lesioned and transplanted rats.

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Correlation of apomorphine- and amphetamine-induced turning with nigrostriatal dopamine content in unilateral 6-hydroxydopamine lesioned rats

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Influence of a single injection of cocaine, amphetamine or GBR 12909 on mRNA expression of striatal neuropeptides

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Neuropathological evidence of graft survival and striatal reinnervation after the transplantation of fetal mesencephalic tissue in a patient with Parkinson's disease

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Three rat preprotachykinin mRNAs encode the neuropeptides substance P and neurokinin A

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Time course of striatal changes induced by 6-hydroxydopamine lesion of the nigrostriatal pathway, as studied by combined evaluation of rotational behaviour and striatal Fos expression.

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7 January 2021

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Identifiers

10.1016/S0006-8993(99)01853-3

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10526102%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10526102%20AND%20SRC:MED&resulttype=core&format=json

1 November 2019

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