(Q48103749)

8 February 2018

title

Early decrease in apurinic/apyrimidinic endonuclease is followed by DNA fragmentation after cold injury-induced brain trauma in mice (English)

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8 February 2018

author name string

Morita-Fujimura Y

1

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8 February 2018

Fujimura M

2

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8 February 2018

Kawase M

3

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8 February 2018

Chan PH

4

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8 February 2018

language of work or name

English

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publication date

1 January 1999

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8 February 2018

published in

Neuroscience

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8 February 2018

volume

93

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8 February 2018

issue

4

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8 February 2018

page(s)

1465-1473

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8 February 2018

Interaction of human apurinic endonuclease and DNA polymerase beta in the base excision repair pathway

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Overexpression of SOD1 in transgenic rats protects vulnerable neurons against ischemic damage after global cerebral ischemia and reperfusion.

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Cold-induced brain edema and infarction are reduced in transgenic mice overexpressing CuZn-Superoxide dismutase

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Experimental brain injury induces regionally distinct apoptosis during the acute and delayed post-traumatic period.

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The enzymology of apurinic/apyrimidinic endonucleases

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7 January 2021

Poly(ADP-ribose) polymerase gene disruption renders mice resistant to cerebral ischemia.

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7 January 2021

Ischemic brain injury is mediated by the activation of poly(ADP-ribose)polymerase

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Early decrease of apurinic/apyrimidinic endonuclease expression after transient focal cerebral ischemia in mice

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Cytosolic redistribution of cytochrome c after transient focal cerebral ischemia in rats.

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Expression of nuclear redox factor ref-1 in the rat hippocampus following global ischemia induced by cardiac arrest

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AP-1 transcriptional activity is regulated by a direct association between thioredoxin and Ref-1

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Attenuation of focal cerebral ischemic injury in transgenic mice overexpressing CuZn superoxide dismutase

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7 January 2021

Reduction of CuZn-superoxide dismutase activity exacerbates neuronal cell injury and edema formation after transient focal cerebral ischemia

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7 January 2021

Temporal profile of in situ DNA fragmentation after transient middle cerebral artery occlusion in the rat

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7 January 2021

Repair of intrinsic DNA lesions

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Mutagenesis by apurinic/apyrimidinic sites

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7 January 2021

Inhibition of interleukin-1beta converting enzyme family proteases (caspases) reduces cold injury-induced brain trauma and DNA fragmentation in mice

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7 January 2021

Occurrence of apoptosis following cold injury-induced brain edema in mice.

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Redox regulation of cellular activation

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7 January 2021

Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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7 January 2021

Developmental expression of APEX nuclease, a multifunctional DNA repair enzyme, in mouse brains

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7 January 2021

Activation of apurinic/apyrimidinic endonuclease in human cells by reactive oxygen species and its correlation with their adaptive response to genotoxicity of free radicals

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7 January 2021

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7 January 2021

DNA strand breakage, activation of poly (ADP-ribose) synthetase, and cellular energy depletion are involved in the cytotoxicity of macrophages and smooth muscle cells exposed to peroxynitrite

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7 January 2021

The caspase family of cysteine proteases

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7 January 2021

Endonuclease activation following focal ischemic injury in the rat brain

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7 January 2021

Identification of residues in the human DNA repair enzyme HAP1 (Ref-1) that are essential for redox regulation of Jun DNA binding

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7 January 2021

Loss of Ref-1 protein expression precedes DNA fragmentation in apoptotic neurons

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7 January 2021

Differential expression of the apurinic / apyrimidinic endonuclease (APE/ref-1) multifunctional DNA base excision repair gene during fetal development and in adult rat brain and testis

1 reference

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7 January 2021

Activation of CPP32-like caspases contributes to neuronal apoptosis and neurological dysfunction after traumatic brain injury.

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7 January 2021

Streptozotocin and alloxan induce DNA strand breaks and poly(ADP–ribose) synthetase in pancreatic islets

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7 January 2021

Nitric oxide activation of poly(ADP-ribose) synthetase in neurotoxicity

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7 January 2021

10.1016/S0306-4522(99)00231-6

Identifiers

DOI

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8 February 2018

1 reference

8 February 2018