(Q48137224)

9 February 2018

title

Bcl-xL is a negative regulator of caspase-3 activation in immature neurons during development. (English)

1 reference

9 February 2018

author name string

Urase K

1

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9 February 2018

Momoi T

2

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9 February 2018

Fujita E

3

1 reference

9 February 2018

Isahara K

4

1 reference

9 February 2018

Uchiyama Y

5

1 reference

9 February 2018

Tokunaga A

6

1 reference

9 February 2018

Nakayama K

7

1 reference

9 February 2018

Motoyama N

8

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9 February 2018

publication date

1 August 1999

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9 February 2018

number of pages

10

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Developmental Brain Research

inferred from page(s)

published in

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9 February 2018

volume

116

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9 February 2018

issue

1

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9 February 2018

page(s)

69-78

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9 February 2018

The Bcl-2 protein family: arbiters of cell survival

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Akt phosphorylation of BAD couples survival signals to the cell-intrinsic death machinery

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X-linked IAP is a direct inhibitor of cell-death proteases

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Function and expression of the Bcl-x gene in the developing and adult nervous system.

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Wortmannin enhances activation of CPP32 (Caspase-3) induced by TNF or anti-Fas

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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7 January 2021

Caenorhabditis elegans gene ced-9 protects cells from programmed cell death

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7 January 2021

Bcl-XL interacts with Apaf-1 and inhibits Apaf-1-dependent caspase-9 activation

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Programmed cell death in animal development

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Detection of activated Caspase-3 by a cleavage site-directed antiserum during naturally occurring DRG neurons apoptosis.

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Decreased apoptosis in the brain and premature lethality in CPP32-deficient mice

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7 January 2021

Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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Bcl-2 gene family in the nervous system

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Peptide inhibitors of the ICE protease family arrest programmed cell death of motoneurons in vivo and in vitro

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7 January 2021

Massive cell death of immature hematopoietic cells and neurons in Bcl-x-deficient mice

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7 January 2021

Specific expression of CPP32 in sensory neurons of mouse embryos and activation of CPP32 in the apoptosis induced by a withdrawal of NGF.

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Identification and inhibition of the ICE/CED-3 protease necessary for mammalian apoptosis

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Developing Caenorhabditis elegans neurons may contain both cell-death protective and killer activities

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Functions of the neurotrophins during nervous system development: what the knockouts are teaching us.

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Caspases: enemies within

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7 January 2021

Detection of activated caspase-3 (CPP32) in the vertebrate nervous system during development by a cleavage site-directed antiserum.

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7 January 2021

Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes

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7 January 2021

The Caenorhabditis elegans genes ced-3 and ced-4 act cell autonomously to cause programmed cell death

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7 January 2021

Serine phosphorylation of death agonist BAD in response to survival factor results in binding to 14-3-3 not BCL-X(L)

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7 January 2021

Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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Identifiers

DOI

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/kbpjlmyvv5fmvglkazrdx2rpci

30 July 2022

mapped directly with Wikidata item

Fatcat ID

release_kbpjlmyvv5fmvglkazrdx2rpci

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Fatcat

https://api.fatcat.wiki/v0/release/kbpjlmyvv5fmvglkazrdx2rpci

30 July 2022

mapped directly with Wikidata item

1 reference

https://api.fatcat.wiki/v0/release/kbpjlmyvv5fmvglkazrdx2rpci

30 July 2022