(Q48172553)

9 February 2018

title

Bcl-2, Bax and Bcl-x expression following kainic acid administration at convulsant doses in the rat. (English)

1 reference

9 February 2018

author name string

López E

1

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9 February 2018

Pozas E

2

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9 February 2018

Rivera R

3

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9 February 2018

Ferrer I

4

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9 February 2018

language of work or name

English

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publication date

1 January 1999

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9 February 2018

published in

Neuroscience

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9 February 2018

volume

91

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9 February 2018

issue

4

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9 February 2018

page(s)

1461-1470

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9 February 2018

Role of Bcl-2 in the brain-derived neurotrophic factor survival response

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Apoptosis repressor genes Bcl-2 and Bcl-x-long are expressed in the rat brain following global ischemia.

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Expression of the apoptosis-effector gene, Bax, is up-regulated in vulnerable hippocampal CA1 neurons following global ischemia

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Cerebral metabolic response to systemic kainic acid: 14-C-deoxyglucose studies

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The Bcl-2 family of proteins, and the regulation of neuronal survival

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BAX is required for neuronal death after trophic factor deprivation and during development

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Bcl-2, Bax, and Bcl-x expression in the CA1 area of the hippocampus following transient forebrain ischemia in the adult gerbil.

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Increased expression of bcl-2 immunoreactivity in the developing cerebral cortex of the rat

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7 January 2021

DNA fragmentation in rat brain after intraperitoneal administration of kainate.

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7 January 2021

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7 January 2021

bcl-x is expressed in embryonic and postnatal neural tissues and functions to prevent neuronal cell death

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7 January 2021

Role of BCL-2 in the survival and function of developing and mature sympathetic neurons

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7 January 2021

Up-regulation of bax and down-regulation of bcl-2 is associated with kainate-induced apoptosis in mouse brain

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7 January 2021

Immunohistochemical detection of Bax and Bcl-2 proteins in gerbil hippocampus following transient forebrain ischemia.

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7 January 2021

Bcl-2 is an inner mitochondrial membrane protein that blocks programmed cell death

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7 January 2021

Cytosol-to-membrane redistribution of Bax and Bcl-X(L) during apoptosis

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7 January 2021

Kainic acid induced limbic seizures: metabolic, behavioral, electroencephalographic and neuropathological correlates

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7 January 2021

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7 January 2021

Bcl-2 gene family in the nervous system

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7 January 2021

Inactivation of bcl-2 results in progressive degeneration of motoneurons, sympathetic and sensory neurons during early postnatal development

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7 January 2021

Bcl-x(S) anatagonizes the protective effects of Bcl-x(L).

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7 January 2021

N-methyl-D-aspartate receptor agonists decrease protooncogene bcl-2 mRNA expression in cultured rat cerebellar granule neurons.

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7 January 2021

Massive cell death of immature hematopoietic cells and neurons in Bcl-x-deficient mice

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7 January 2021

Targeted disruption of Bcl-2 alpha beta in mice: occurrence of gray hair, polycystic kidney disease, and lymphocytopenia

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7 January 2021

Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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7 January 2021

Kainate-induced apoptotic cell death in hippocampal neurons.

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7 January 2021

Evidence for apoptotic cell death in Huntington disease and excitotoxic animal models.

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7 January 2021

Bcl-2 and the regulation of programmed cell death

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7 January 2021

Apoptosis of bcl-x-deficient telencephalic cells in vitro.

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7 January 2021

Activation of hippocampal metabotropic excitatory amino acid receptors leads to seizures and neuronal damage

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7 January 2021

Interactions among members of the Bcl-2 protein family analyzed with a yeast two-hybrid system.

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7 January 2021

Widespread patterns of neuronal damage following systemic or intracerebral injections of kainic acid: a histological study

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7 January 2021

Multiple Bcl-2 family members demonstrate selective dimerizations with Bax

1 reference

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7 January 2021

Kainic acid induced seizures: neurochemical and histopathological changes

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7 January 2021

Kainic acid-induced seizures: dose-relationship of behavioural, neurochemical and histopathological changes.

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https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

The biochemical mechanisms of the excitotoxicity of kainic acid. Free radical formation

1 reference

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7 January 2021

Bcl-2-deficient mice demonstrate fulminant lymphoid apoptosis, polycystic kidneys, and hypopigmented hair

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

Excitatory Amino Acid Transmitters

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

Cloning of a putative high-affinity kainate receptor expressed predominantly in hippocampal CA3 cells

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

Proapoptotic protein Bax heterodimerizes with Bcl-2 and homodimerizes with Bax via a novel domain (BH3) distinct from BH1 and BH2

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

BCL-2 blocks glutamate toxicity in neural cell lines

1 reference

https://api.crossref.org/works/10.1016%2FS0306-4522%2898%2900704-0

7 January 2021

10.1016/S0306-4522(98)00704-0

Identifiers

DOI

1 reference

9 February 2018

PubMed ID

10391451

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9 February 2018