(Q48183597)

9 February 2018

title

Amyloidosis in transgenic mice expressing murine amyloidogenic apolipoprotein A-II (Apoa2c). (English)

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9 February 2018

author name string

Fengxia Ge

1

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9 February 2018

Junjie Yao

2

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9 February 2018

Xiaoying Fu

3

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9 February 2018

Zhanjun Guo

4

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9 February 2018

Jingmin Yan

5

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9 February 2018

Beiru Zhang

6

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9 February 2018

Huanyu Zhang

7

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9 February 2018

Hiroshi Tomozawa

8

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9 February 2018

Junichi Miyazaki

9

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9 February 2018

Jinko Sawashita

10

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9 February 2018

Masayuki Mori

11

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9 February 2018

Keiichi Higuchi

12

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9 February 2018

publication date

30 April 2007

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9 February 2018

published in

Laboratory Investigation

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9 February 2018

volume

87

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9 February 2018

issue

7

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9 February 2018

page(s)

633-643

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9 February 2018

Polymorphism of apolipoprotein A-II (apoA-II) among inbred strains of mice. Relationship between the molecular type of apoA-II and mouse senile amyloidosis

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Polymorphisms of mouse apolipoprotein A-II: seven alleles found among 41 inbred strains of mice

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Tissue distribution, biochemical properties, and transmission of mouse type A AApoAII amyloid fibrils

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Mouse senile amyloid deposition is suppressed by adenovirus-mediated overexpression of amyloid-resistant apolipoprotein A-II

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Regulation of the metabolism of plasma lipoproteins by apolipoprotein A-II

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Extrahepatic expression of apolipoprotein A-II in mouse tissues: possible contribution to mouse senile amyloidosis

21 January 2018

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Folding proteins in fatal ways

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7 January 2021

Amyloid deposits and amyloidosis. The beta-fibrilloses (first of two parts).

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7 January 2021

Molecular mechanisms of amyloidosis

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7 January 2021

Accelerated senile amyloidosis induced by amyloidogenic Apoa-II gene shortens the life span of mice but does not accelerate the rate of senescence

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7 January 2021

Wild type ApoA-II gene does not rescue senescence-accelerated mouse (SAMP1) from short life span and accelerated mortality

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7 January 2021

Transmission of mouse senile amyloidosis

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7 January 2021

Induction of protein conformational change in mouse senile amyloidosis

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7 January 2021

Induction of AApoAII amyloidosis by various heterogeneous amyloid fibrils

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7 January 2021

Transmissibility of systemic amyloidosis by a prion-like mechanism

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7 January 2021

Acceleration of murine AA amyloidosis by oral administration of amyloid fibrils extracted from different species

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7 January 2021

Amyloid fibril proteins

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7 January 2021

Amyloid fibril formation of alpha-synuclein is accelerated by preformed amyloid seeds of other proteins: implications for the mechanism of transmissible conformational diseases

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7 January 2021

Seeding specificity in amyloid growth induced by heterologous fibrils

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7 January 2021

Mechanism of cross-species prion transmission: an infectious conformation compatible with two highly divergent yeast prion proteins

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7 January 2021

Efficient selection for high-expression transfectants with a novel eukaryotic vector

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7 January 2021

Sensitive detection of pathological prion protein by cyclic amplification of protein misfolding

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7 January 2021

Tricine-sodium dodecyl sulfate-polyacrylamide gel electrophoresis for the separation of proteins in the range from 1 to 100 kDa

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7 January 2021

Development of congenic strains of mice carrying amyloidogenic apolipoprotein A-II (Apoa2c). Apoa2c reduces the plasma level and the size of high density lipoprotein

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7 January 2021

Physical, chemical, and ultrastructural studies of water-soluble human amyloid fibrils. Comparative analyses of nine amyloid preparations

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7 January 2021

Therapeutic approaches to protein-misfolding diseases

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7 January 2021

The biological and chemical basis for tissue-selective amyloid disease

1 reference

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7 January 2021

Atherosclerosis in transgenic mice overexpressing apolipoprotein A-II.

1 reference

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7 January 2021

Dietary fat modulation of apoA-II metabolism and prevention of senile amyloidosis in the senescence- accelerated mouse

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Amyloidosis Modifier Genes in the Less Amyloidogenic A/J Mouse Strain

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7 January 2021

Antibodies to a scrapie prion protein

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7 January 2021

Identification of prion amyloid filaments in scrapie-infected brain

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7 January 2021

Anchorless prion protein results in infectious amyloid disease without clinical scrapie

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7 January 2021

The most infectious prion protein particles.

1 reference

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7 January 2021

Cell-free formation of protease-resistant prion protein.

1 reference

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7 January 2021

Support for the prion hypothesis for inheritance of a phenotypic trait in yeast

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Propagation of the yeast prion-like [psi+] determinant is mediated by oligomerization of the SUP35-encoded polypeptide chain release factor

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Prion propagation in mice expressing human and chimeric PrP transgenes implicates the interaction of cellular PrP with another protein

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Instability, unfolding and aggregation of human lysozyme variants underlying amyloid fibrillogenesis

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Alternative conformations of amyloidogenic proteins govern their behavior

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

The acid-mediated denaturation pathway of transthyretin yields a conformational intermediate that can self-assemble into amyloid

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

Transmissibility of mouse AApoAII amyloid fibrils: inactivation by physical and chemical methods.

1 reference

https://api.crossref.org/works/10.1038%2FLABINVEST.3700559

7 January 2021

10.1038/LABINVEST.3700559

Identifiers

DOI

1 reference

9 February 2018

1 reference

9 February 2018