(Q48216036)

9 February 2018

title

Loss of insulin receptor immunoreactivity from the substantia nigra pars compacta neurons in Parkinson's disease. (English)

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9 February 2018

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4

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9 February 2018

author name string

I Moroo

1

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9 February 2018

T Yamada

2

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9 February 2018

H Makino

3

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9 February 2018

P L McGeer

5

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9 February 2018

E G McGeer

6

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9 February 2018

K Hirayama

7

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9 February 2018

publication date

1 January 1994

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9 February 2018

published in

Acta Neuropathologica

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9 February 2018

volume

87

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9 February 2018

page(s)

343-348

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9 February 2018

issue

4

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9 February 2018

https://scigraph.springernature.com/pub.10.1007/bf00313602

exact match

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cites work

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Human insulin receptor and its relationship to the tyrosine kinase family of oncogenes

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Loss of basic fibroblast growth factor in substantia nigra neurons in Parkinson's disease.

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A unifying hypothesis for the cause of amyotrophic lateral sclerosis, parkinsonism, and alzheimer disease

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12 December 2020

Immunohistochemical localization of insulin receptors and phosphotyrosine in the brainstem of the adult rat

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BDNF is a neurotrophic factor for dopaminergic neurons of the substantia nigra.

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Development of brain insulin receptors

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Insulin protects brain tissue against focal ischemia in rats

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Cortical astrocytes activated by basic fibroblast growth factor secrete molecules that stimulate differentiation of mesencephalic dopaminergic neurons

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Insulin stimulates macromolecular synthesis in cultured glial cells from rat brain.

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Regulation of food intake and body weight by insulin.

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Effect of insulin on central catecholamines

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Insulin receptors and insulin modulation of norepinephrine uptake in neuronal cultures from rat brain.

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Brain-derived neurotrophic factor protects dopamine neurons against 6-hydroxydopamine and N-methyl-4-phenylpyridinium ion toxicity: involvement of the glutathione system.

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Neurotrophic factors and Parkinson's disease

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Autoradiographic localization of insulin receptors in rat brain: prominence in olfactory and limbic areas.

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Identifiers

DOI

10.1007/BF00313602

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9 February 2018

PubMed ID

8017169

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9 February 2018