(Q48253391)

10 February 2018

title

Unilateral dopamine depletion paradoxically enhances amphetamine-induced Fos expression in basal ganglia output structures. (English)

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10 February 2018

author name string

Wirtshafter D

series ordinal

1

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10 February 2018

Asin KE

series ordinal

2

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10 February 2018

language of work or name

English

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publication date

1 April 1999

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10 February 2018

published in

Brain Research

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10 February 2018

volume

824

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10 February 2018

issue

1

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10 February 2018

page(s)

81-88

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10 February 2018

Turning behaviour and catalepsy after injection of excitatory amino acids into rat substantia nigra

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Reduction of apomorphine-induced rotational behaviour by subthalamic lesion in 6-OHDA lesioned rats is associated with a normalization of firing rate and discharge pattern of pars reticulata neurons

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The neurons of the substantia nigra and zona incerta which project to the cat superior colliculus are GABA immunoreactive: a double-label study using GABA immunocytochemistry and lectin retrograde transport

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Coordinate suppression of striatal ngfi-a and c-fos produces locomotor asymmetry and up-regulation of IEGs in the globus pallidus

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Destruction of the nigrostriatal pathway increases Fos-like immunoreactivity predominantly in striatopallidal neurons.

7 January 2021

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Characterization of dopamine receptor binding sites in the subthalamic nucleus

7 January 2021

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Apomorphine and dopamine D(1) receptor agonists increase the firing rates of subthalamic nucleus neurons

7 January 2021

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Rapid development of D1 and D2 dopamine receptor supersensitivity as indicated by striatal and pallidal Fos expression

7 January 2021

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L-Glutamic Acid Decarboxylase in Parkinson's Disease: Effect of L-Dopa Therapy

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Dopamine D2 receptor control of pallidal fos expression: comparisons between intact and 6-hydroxydopamine-treated hemispheres

7 January 2021

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7 January 2021

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Amphetamine-induced increase in motor activity is correlated with higher firing rates of non-dopamine neurons in substantia nigra and ventral tegmental area

7 January 2021

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The effects of striatal lesion on turning behavior and globus pallidus single unit response to dopamine agonist administration

7 January 2021

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Genetics, haloperidol, and the Fos response in the basal ganglia: a comparison of the C57BL/6J and DBA/2J inbred mouse strains

7 January 2021

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D1-like and D2-like dopamine receptors synergistically activate rotation and c-fos expression in the dopamine-depleted striatum in a rat model of Parkinson's disease

7 January 2021

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Striatonigral projection neurons contain D1 dopamine receptor-activated c-fos.

7 January 2021

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D1 and D2 dopamine receptors differentially regulate c-fos expression in striatonigral and striatopallidal neurons

7 January 2021

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Acute monoaminergic depletion in the rat potentiates the excitatory effect of the subthalamic nucleus in the substantia nigra pars reticulata but not in the pallidal complex

7 January 2021

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Distinct commissural pathways are involved in the enhanced release of dopamine induced in the contralateral caudate nucleus and substantia nigra by unilateral application of GABA in the cat thalamic motor nuclei.

7 January 2021

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7 January 2021

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D1 dopamine receptors influence Fos immunoreactivity in the globus pallidus and subthalamic nucleus of intact and nigrostriatal-lesioned rats.

7 January 2021

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Differing influences of dopamine agonists and antagonists on Fos expression in identified populations of globus pallidus neurons

7 January 2021

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Subthalamic nucleus and globus pallidus lesions alter activity in nigrothalamic neurons in rats.

7 January 2021

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Expression of c-fos protein in brain: metabolic mapping at the cellular level

7 January 2021

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Effect of electrical stimulation of the cerebral cortex on the expression of the Fos protein in the basal ganglia

7 January 2021

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Neurophysiological properties of pallidal neurons in Parkinson's disease

7 January 2021

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Role of the corpus callosum in expression of behavioral asymmetries induced by a unilateral dopamine lesion of the substantia nigra in the rat.

7 January 2021

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Substantia nigra and motor control in the rat: effect of intranigral alpha-kainate and gamma-D-glutamylaminomethylsulphonate on motility.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Amphetamine-induced Fos expression in globus pallidus is altered by frontal cortex injury

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Mapping of the motor pathways in rats: c-fos induction by intracortical microstimulation of the motor cortex correlated with efferent connectivity of the site of cortical stimulation.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Effect of dopamine system activation on substantia nigra pars reticulata output neurons: variable single-unit responses in normal rats and inhibition in 6-hydroxydopamine-lesioned rats.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Interactive effects of stimulation of D1 and D2 dopamine receptors on fos-like immunoreactivity in the normosensitive rat striatum.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Dopamine antagonists induce fos-like-immunoreactivity in the substantia nigra and entopeduncular nucleus of the rat.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Dopamine agonists and stress produce different patterns of Fos-like immunoreactivity in the lateral habenula

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Interactive effects of stimulation of D1 and D2 dopamine receptors on Fos expression in the lateral habenula.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

Expression of the Immediate Early Gene c-fos in Basal Ganglia: Induction by Dopaminergic Drugs

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2899%2901091-4

inferred from DOI database lookup

7 January 2021

based on heuristic

Identifiers

DOI

10.1016/S0006-8993(99)01091-4

1 reference

10 February 2018

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10 February 2018