(Q48312558)

10 February 2018

title

Astrocytes within multiple sclerosis lesions upregulate sodium channel Nav1.5. (English)

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10 February 2018

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Joel A Black

1

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10 February 2018

Jia Newcombe

series ordinal

2

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10 February 2018

Stephen G Waxman

series ordinal

3

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10 February 2018

language of work or name

English

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publication date

10 February 2010

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10 February 2018

published in

Brain

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10 February 2018

volume

133

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10 February 2018

issue

Pt 3

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10 February 2018

page(s)

835-846

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10 February 2018

A tetrodotoxin-resistant voltage-gated sodium channel expressed by sensory neurons

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NaN, a novel voltage-gated Na channel, is expressed preferentially in peripheral sensory neurons and down-regulated after axotomy

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Primary motor neurons fail to up-regulate voltage-gated sodium channel Na(v)1.3/brain type III following axotomy resulting from spinal cord injury

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Identification of PN1, a predominant voltage-dependent sodium channel expressed principally in peripheral neurons

21 January 2018

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Expression of MCP-1 by reactive astrocytes in demyelinating multiple sclerosis lesions

21 January 2018

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Phagocytosis of myelin by astrocytes in explants of adult rabbit cerebral white matter maintained on Gelfoam matrix

21 January 2018

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Mechanisms of disease: sodium channels and neuroprotection in multiple sclerosis-current status

21 January 2018

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Involvement of Kv1.1 and Nav1.5 in proliferation of gastric epithelial cells

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21 January 2018

Identifiers

DOI

10.1093/BRAIN/AWQ003

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10 February 2018

1 reference

10 February 2018