(Q48345720)

English

C-terminal fragments of amyloid-beta peptide cause cholinergic axonal degeneration by a toxic effect rather than by physical injury in the nondemented human brain.

scientific article published in April 2003

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

C-terminal fragments of amyloid-beta peptide cause cholinergic axonal degeneration by a toxic effect rather than by physical injury in the nondemented human brain. (English)

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Frédéric Checler

5

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

author name string

Peter Kasa

1

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Henrietta Papp

2

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Janos Zombori

3

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Peter Mayer

4

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

publication date

1 April 2003

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Neurochemical Research

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

28

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

3-4

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

493-498

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

https://scigraph.springernature.com/pub.10.1023/a:1022813104905

0 references

Visualization of detailed acetylcholinesterase fiber and neuron staining in rat brain by a sensitive histochemical procedure.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The cause of neuronal degeneration in Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Senile plaques exert no mass lesion effect on surrounding neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Evidence that transmitter-containing dystrophic neurites precede those containing paired helical filaments within senile plaques in the entorhinal cortex of nondemented elderly and Alzheimer's disease patients.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Hypothesis: amyloid beta-peptides truncated at the N-terminus contribute to the pathogenesis of Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Human amyloid-beta1-42 applied in vivo inhibits the fast axonal transport of proteins in the sciatic nerve of rat.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The nonfibrillar amyloid beta-peptide induces apoptotic neuronal cell death: involvement of its C-terminal fusogenic domain.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Staging of Alzheimer's disease-related neurofibrillary changes

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Cholinergic fibre loss associated with diffuse plaques in the non-demented elderly: the preclinical stage of Alzheimer's disease?

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Neuritic pathology and dementia in Alzheimer's disease.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Dystrophic neurite formation associated with age-related beta amyloid deposition in the neocortex: clues to the genesis of neurofibrillary pathology.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Selective phosphorylation of adult tau isoforms in mature hippocampal neurons exposed to fibrillar A beta.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Plaque-induced neurite abnormalities: implications for disruption of neural networks in Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

beta-Amyloid induces neuritic dystrophy in vitro: similarities with Alzheimer pathology

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Two types of dystrophic neurites in senile plaques of Alzheimer disease and elderly non-demented cases.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Regulation of tau phosphorylation in Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Donepezil dose-dependently inhibits acetylcholinesterase activity in various areas and in the presynaptic cholinergic and the postsynaptic cholinoceptive enzyme-positive structures in the human and rat brain

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Early phosphorylation of tau in Alzheimer's disease occurs at Ser-202 and is preferentially located within neurites.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Characterization of new polyclonal antibodies specific for 40 and 42 amino acid-long amyloid beta peptides: their use to examine the cell biology of presenilins and the immunohistochemistry of sporadic Alzheimer's disease and cerebral amyloid angiop

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

beta-amyloid fibrils induce tau phosphorylation and loss of microtubule binding.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

N-terminus truncated beta-amyloid peptides and C-terminus truncated secreted forms of amyloid precursor protein: distinct roles in the pathogenesis of Alzheimer's disease.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Plaque biogenesis in brain aging and Alzheimer's disease. I. Progressive changes in phosphorylation states of paired helical filaments and neurofilaments

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Neuropathological criteria for the diagnosis of Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

The cholinergic system in Alzheimer's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Amyloid beta peptide membrane perturbation is the basis for its biological effects

1 reference

https://pubmed.ncbi.nlm.nih.gov/12675137

12 December 2020

based on heuristic

inferred from PubMed ID database lookup

Identifiers

10.1023/A:1022813104905

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

Dimensions Publication ID

0 references

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/12675137

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