(Q48349921)

10 February 2018

title

Effect of IL-1alpha on the release of norepinephrine in rat hypothalamus. (English)

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10 February 2018

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Kaur D

1

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10 February 2018

Cruess DF

series ordinal

2

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10 February 2018

Potter WZ

series ordinal

3

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10 February 2018

publication date

1 October 1998

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10 February 2018

Journal of Neuroimmunology

published in

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10 February 2018

volume

90

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10 February 2018

issue

2

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10 February 2018

page(s)

122-127

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10 February 2018

Bidirectional transport of interleukin-1 alpha across the blood-brain barrier

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Central activation of thermogenesis and fever by interleukin-1 beta and interleukin-1 alpha involves different mechanisms

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The cell surface receptors for interleukin-1 alpha and interleukin-1 beta are identical

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Systemic interleukin-1 administration stimulates hypothalamic norepinephrine metabolism parallelling the increased plasma corticosterone.

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Interleukin-1 induces changes in norepinephrine metabolism in the rat brain

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Peripheral administration of interleukin-1 increases extracellular concentrations of norepinephrine in rat hypothalamus: comparison with plasma corticosterone

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Interleukin-1 directly stimulates the release of corticotrophin releasing factor from rat hypothalamus

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7 January 2021

Interleukin-1 stimulates ACTH release by an indirect action which requires endogenous corticotropin releasing factor.

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Corticotropin-releasing factor activates noradrenergic neurons of the locus coeruleus.

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Identifiers

DOI

1 reference

10 February 2018

PubMed ID

9817439

1 reference

10 February 2018