(Q48354077)

English

Amyloid beta protein inhibits cellular MTT reduction not by suppression of mitochondrial succinate dehydrogenase but by acceleration of MTT formazan exocytosis in cultured rat cortical astrocytes.

scientific article published in August 1998

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

Amyloid beta protein inhibits cellular MTT reduction not by suppression of mitochondrial succinate dehydrogenase but by acceleration of MTT formazan exocytosis in cultured rat cortical astrocytes. (English)

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

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1 reference

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author name string

Abe K

1

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

Saito H

2

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

publication date

1 August 1998

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

Neuroscience Research

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

31

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

4

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

295-305

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

Menadione toxicity in cultured rat cortical astrocytes.

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Human amylin mimics amyloid beta protein-induced reactive gliosis and inhibition of cellular redox activity in cultured astrocytes.

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Congo red reverses amyloid beta protein-induced cellular stress in astrocytes.

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3-Nitropropionate, the toxic substance of Indigofera , is a suicide inactivator of succinate dehydrogenase

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Isolation and sequence determination of rat islet amyloid polypeptide.

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Syntheses, structures and anorectic effects of human and rat amylin

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Hydrogen peroxide mediates amyloid beta protein toxicity

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beta-Amyloid of Alzheimer's disease induces reactive gliosis that inhibits axonal outgrowth.

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Purification and characterization of a peptide from amyloid-rich pancreases of type 2 diabetic patients

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Astrocytes protect neurons from hydrogen peroxide toxicity

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Glial fibrillary acidic protein and Alzheimer-type senile dementia.

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Synthesis and secretion of nerve growth factor by mouse astroglial cells in culture.

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In vitro neurite extension by granule neurons is dependent upon astroglial-derived fibroblast growth factor.

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Nanomolar amyloid beta protein-induced inhibition of cellular redox activity in cultured astrocytes

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Mechanism of cellular 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction.

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Cytotoxic amyloid peptides inhibit cellular 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) reduction by enhancing MTT formazan exocytosis.

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Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red.

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Astrocytes and the plaques of Alzheimer's disease

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The extending astroglial process: development of glial cell shape, the growing tip, and interactions with neurons

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Calcium-destabilizing and neurodegenerative effects of aggregated beta-amyloid peptide are attenuated by basic FGF.

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Neurotoxicity of human amylin in rat primary hippocampal cultures: similarity to Alzheimer's disease amyloid-beta neurotoxicity

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Mechanisms of cell death induced by the mitochondrial toxin 3-nitropropionic acid: acute excitotoxic necrosis and delayed apoptosis.

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beta-Amyloid-mediated inhibition of redox activity (MTT reduction) is not an indicator of astroglial degeneration.

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In vitro aging of beta-amyloid protein causes peptide aggregation and neurotoxicity

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Beta-amyloid-induced changes in cultured astrocytes parallel reactive astrocytosis associated with senile plaques in Alzheimer's disease

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Neuron-glia relationship during granule cell migration in developing cerebellar cortex. A Golgi and electronmicroscopic study in Macacus Rhesus

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Inhibition of PC12 cell redox activity is a specific, early indicator of the mechanism of beta-amyloid-mediated cell death

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The intracellular component of cellular 3-(4,5-dimethylthiazol-2-yl)-2, 5-diphenyltetrazolium bromide (MTT) reduction is specifically inhibited by beta-amyloid peptides.

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STUDIES ON SUCCINATE-TETRAZOLIUM REDUCTASE SYSTEMS. III. POINTS OF COUPLING OF FOUR DIFFERENT TETRAZOLIUM SALTS

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Do astrocytes process neural information?

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Cell death in primary cultures of mouse neurons and astrocytes during exposure to and 'recovery' from hypoxia, substrate deprivation and simulated ischemia

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Astrocytes protect cultured neurons from degeneration induced by anoxia

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Amyloid fibrils in human insulinoma and islets of Langerhans of the diabetic cat are derived from a neuropeptide-like protein also present in normal islet cells

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Islet amyloid polypeptide: pinpointing amino acid residues linked to amyloid fibril formation

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RAGE and amyloid-beta peptide neurotoxicity in Alzheimer's disease

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Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

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Hypoxia-induced dysfunctions and injury of astrocytes in primary cell cultures

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Identifiers

10.1016/S0168-0102(98)00055-8

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

10 February 2018

http://europepmc.org/abstract/MED/9809588

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