(Q48401390)

11 February 2018

title

Anti-high mobility group box-1 antibody therapy for traumatic brain injury (English)

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Hideo Kohka Takahashi

14

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author name string

Yu Okuma

1

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Keyue Liu

2

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Hidenori Wake

3

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Jiyong Zhang

4

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Tomoko Maruo

5

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Isao Date

6

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Tadashi Yoshino

7

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Aiji Ohtsuka

8

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Naoki Otani

9

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Satoshi Tomura

10

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Katsuji Shima

11

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Yasuhiko Yamamoto

12

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Hiroshi Yamamoto

13

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Shuji Mori

15

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Masahiro Nishibori

16

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publication date

22 August 2012

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published in

Annals of Neurology

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volume

72

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issue

3

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page(s)

373-384

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Blood-brain barrier breakdown as a therapeutic target in traumatic brain injury

cites work

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The duality of the inflammatory response to traumatic brain injury

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High-mobility group box 1 protein (HMGB1): nuclear weapon in the immune arsenal

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The HMGB1 receptor RAGE mediates ischemic brain damage

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Toll-like receptor 4 and high-mobility group box-1 are involved in ictogenesis and can be targeted to reduce seizures

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High-mobility group box 1 is involved in the initial events of early loss of transplanted islets in mice

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Anti-high mobility group box 1 monoclonal antibody ameliorates brain infarction induced by transient ischemia in rats

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Anti-high mobility group box-1 monoclonal antibody protects the blood-brain barrier from ischemia-induced disruption in rats.

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Temporal and spatial profile of phosphorylated mitogen-activated protein kinase pathways after lateral fluid percussion injury in the cortex of the rat brain

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RAGE control of diabetic nephropathy in a mouse model: effects of RAGE gene disruption and administration of low-molecular weight heparin

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Early release of HMGB-1 from neurons after the onset of brain ischemia

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Early microvascular and neuronal consequences of traumatic brain injury: a light and electron microscopic study in rats

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The role of hypoxia-inducible factor-1α, aquaporin-4, and matrix metalloproteinase-9 in blood-brain barrier disruption and brain edema after traumatic brain injury

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Administration of S-nitrosoglutathione after traumatic brain injury protects the neurovascular unit and reduces secondary injury in a rat model of controlled cortical impact

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Matrix metalloproteinase-mediated disruption of tight junction proteins in cerebral vessels is reversed by synthetic matrix metalloproteinase inhibitor in focal ischemia in rat.

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1400W, a potent selective inducible NOS inhibitor, improves histopathological outcome following traumatic brain injury in rats

21 January 2018

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The blood-brain barrier in brain homeostasis and neurological diseases

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Depression and cognitive complaints following mild traumatic brain injury

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Identifiers

DOI

10.1002/ANA.23602

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11 February 2018

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11 February 2018