(Q48403494)

11 February 2018

title

Evidence of increased excitability in GEPR hippocampus preceding development of seizure susceptibility. (English)

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11 February 2018

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Verma-Ahuja S

1

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11 February 2018

Evans MS

series ordinal

2

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11 February 2018

Espinosa JA

series ordinal

3

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11 February 2018

publication date

1 August 1998

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11 February 2018

number of pages

13

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inferred from page(s)

published in

Epilepsy Research

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11 February 2018

volume

31

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11 February 2018

issue

3

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11 February 2018

page(s)

161-173

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11 February 2018

Convulsive status epilepticus in infants and children. A study of 239 cases

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Epileptiform burst afterhyperolarization: calcium-dependent potassium potential in hippocampal CA1 pyramidal cells

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Genesis of epileptic interictal spikes. New knowledge of cortical feedback systems suggests a neurophysiological explanation of brief paroxysms

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Neuroanatomical localization of structures responsible for seizures in the GEPR: lesion studies

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Effect of midbrain and pontine tegmental lesions on audiogenic seizures in genetically epilepsy-prone rats

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Loss of synaptic inhibition during repetitive stimulation in genetically epilepsy-prone rats (GEPR).

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Developing genetically epilepsy-prone rats have an abnormal seizure response to flurothyl

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Verapamil blocks the afterhyperpolarization but not the spike frequency accommodation of rat CA1 pyramidal cells in vitro

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Calcium-dependent current generating the afterhyperpolarization of hippocampal neurons.

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Noradrenaline blocks accommodation of pyramidal cell discharge in the hippocampus

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Increased seizure susceptibility of the immature brain

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Depression of the fast IPSP underlies paired-pulse facilitation in area CA1 of the rat hippocampus

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Potassium currents in hippocampal pyramidal cells

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Evidence for decreased calcium dependent potassium conductance in hippocampal CA3 neurons of genetically epilepsy-prone rats

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Noradrenergic terminal fields as determinants of seizure predisposition in GEPR-3s: a neuroanatomic assessment with intracerebral microinjections of 6-hydroxydopamine

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Thalamic deficiency in norepinephrine release detected via intracerebral microdialysis: a synaptic determinant of seizure predisposition in the genetically epilepsy-prone rat.

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Norepinephrine-stimulated phosphatidylinositol metabolism in genetically epilepsy-prone and kindled rats

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7 January 2021

10.1016/S0920-1211(98)00027-8

Identifiers

DOI

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11 February 2018

1 reference

11 February 2018