(Q48494490)

12 February 2018

title

Changes in blood-brain barrier permeability following neurotoxic lesions of rat brain can be visualised with trypan blue (English)

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12 February 2018

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D S Reynolds

1

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12 February 2018

A J Morton

series ordinal

2

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12 February 2018

publication date

1 January 1998

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12 February 2018

Journal of Neuroscience Methods

published in

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12 February 2018

volume

79

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12 February 2018

issue

1

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12 February 2018

page(s)

115-121

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12 February 2018

Replication of the neurochemical characteristics of Huntington's disease by quinolinic acid

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Serum proteins bypass the blood-brain fluid barriers for extracellular entry to the central nervous system

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Lesion of striatal neurons with kainic acid provides a model for Huntington's chorea

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Intraventricular infusion of N-methyl-D-aspartate. 1. Acute blood-brain barrier consequences

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The blood-brain barrier

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Nature and distribution of brain lesions in rats intoxicated with 3-nitropropionic acid: a type of hypoxic (energy deficient) brain damage

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Correlation of morphologic brain lesions with physiologic alterations and blood-brain barrier impairment in 3-nitropropionic acid toxicity in rats

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Complement activation in the central nervous system following blood-brain barrier damage in man.

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NMDA- and endothelin-1-induced increases in blood-brain barrier permeability quantitated with Lucifer yellow.

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Chronically administered 3-nitropropionic acid induces striatal lesions attributed to dysfunction of the blood-brain barrier

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Acute 3-nitropropionic acid intoxication induces striatal astrocytic cell death and dysfunction of the blood-brain barrier: involvement of dopamine toxicity

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The relationship between plasma protein extravasation and remote tissue changes after experimental brain infarction

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Ischemic cerebral edema and the blood-brain barrier. Distributions of pertechnetate, albumin, sodium, and antipyrine in brains of cats after occlusion of the middle cerebral artery

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The blood-brain barrier to protein tracers in focal cerebral ischemia and infarction caused by occlusion of the middle cerebral artery

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Edema and vascular permeability in cerebral ischemia: comparison between ischemic neuronal damage and infarction

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7 January 2021

Early and transient increase of rat hippocampal blood-brain barrier permeability to amino acids during kainic acid-induced seizures.

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Fine structural localization of a blood-brain barrier to exogenous peroxidase

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Widespread patterns of neuronal damage following systemic or intracerebral injections of kainic acid: a histological study

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A histological study of kainic acid-induced lesions in the rat brain

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7 January 2021

Local and distant neuronal degeneration following intrastriatal injection of kainic acid.

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Blood-brain barrier changes with kainic acid-induced limbic seizures.

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7 January 2021

10.1016/S0165-0270(97)00168-4

Identifiers

DOI

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12 February 2018

1 reference

12 February 2018