(Q48576196)

12 February 2018

title

Inhibition of striatal energy metabolism produces cell loss in the ipsilateral substantia nigra. (English)

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12 February 2018

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Sonsalla PK

1

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12 February 2018

Manzino L

2

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12 February 2018

Sinton CM

3

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12 February 2018

Liang CL

4

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12 February 2018

German DC

5

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12 February 2018

Zeevalk GD

6

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12 February 2018

language of work or name

English

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publication date

1 October 1997

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12 February 2018

number of pages

4

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based on heuristic

inferred from page(s)

published in

Brain Research

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12 February 2018

volume

773

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12 February 2018

issue

1-2

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12 February 2018

page(s)

223-226

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12 February 2018

Damage to dopaminergic nerve terminals in mice by combined treatment of intrastriatal malonate with systemic methamphetamine or MPTP

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Neurochemical and histologic characterization of striatal excitotoxic lesions produced by the mitochondrial toxin 3-nitropropionic acid

7 January 2021

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Electron transfer complexes I and IV of platelets are abnormal in Parkinson's disease but normal in Parkinson-plus syndromes.

7 January 2021

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Respiratory chain abnormalities in skeletal muscle from patients with Parkinson's disease.

7 January 2021

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Age-dependent vulnerability of the striatum to the mitochondrial toxin 3-nitropropionic acid.

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

The neurotoxin MPTP causes degeneration of specific nucleus A8, A9 and A10 dopaminergic neurons in the mouse

7 January 2021

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Enzymatic oxidation of dopamine: the role of prostaglandin H synthase

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Platelet mitochondrial function in Parkinson's disease. The Royal Kings and Queens Parkinson Disease Research Group

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

The high sensitivity to rotenone of striatal dopamine uptake suggests the existence of a constitutive metabolic deficiency in dopaminergic neurons from the substantia nigra.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

A selective toxicity toward cultured mesencephalic dopaminergic neurons is induced by the synergistic effects of energetic metabolism impairment and NMDA receptor activation.

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

An immunohistochemical study on alpha-ketoglutarate dehydrogenase complex in Parkinson's disease

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Progressive degeneration of nigrostriatal dopamine neurons following intrastriatal terminal lesions with 6-hydroxydopamine: a combined retrograde tracing and immunocytochemical study in the rat.

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Origin and functional consequences of the complex I defect in Parkinson's disease

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

Oxygen radical mechanisms of brain injury following ischemia and reperfusion

7 January 2021

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https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

In vivo vulnerability of dopamine neurons to inhibition of energy metabolism.

7 January 2021

1 reference

https://api.crossref.org/works/10.1016%2FS0006-8993%2897%2900941-4

10.1016/S0006-8993(97)00941-4

7 January 2021

Identifiers

DOI

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12 February 2018

1 reference

12 February 2018