(Q48580228)

13 February 2018

title

N-acetylaspartylglutamate (NAAG) protects against rat striatal quinolinic acid lesions in vivo (English)

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13 February 2018

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3

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13 February 2018

author name string

L R Orlando

1

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13 February 2018

R Luthi-Carter

2

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13 February 2018

J T Coyle

4

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13 February 2018

J B Penney

5

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13 February 2018

A B Young

6

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13 February 2018

language of work or name

English

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publication date

1 October 1997

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13 February 2018

number of pages

4

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inferred from page(s)

published in

Neuroscience Letters

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13 February 2018

volume

236

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13 February 2018

issue

2

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13 February 2018

page(s)

91-94

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13 February 2018

Replication of the neurochemical characteristics of Huntington's disease by quinolinic acid

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Activation of class II or III metabotropic glutamate receptors protects cultured cortical neurons against excitotoxic degeneration

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The inhibitory mGluR agonist, S-4-carboxy-3-hydroxy-phenylglycine selectively attenuates NMDA neurotoxicity and oxygen-glucose deprivation-induced neuronal death

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N-acetylaspartylglutamate selectively inhibits neuronal responses to N-methyl-D-aspartic acid in vitro

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N-acetyl-aspartyl-glutamate (NAAG) elicits rapid increase in intraneuronal Ca2+ in vitro

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Molecular diversity of glutamate receptors and implications for brain function

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Metabotropic receptors in excitotoxicity: (S)-4-carboxy-3-hydroxyphenylglycine ((S)-4C3HPG) protects against rat striatal quinolinic acid lesions

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Interactions Between N‐Acetylaspartylglutamate and AMPA, Kainate, and NMDA Binding Sites

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High concentrations of N-acetylaspartylglutamate (NAAG) selectively activate NMDA receptors on mouse spinal cord neurons in cell culture

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N-acetylaspartylglutamate inhibits forskolin-stimulated cyclic AMP levels via a metabotropic glutamate receptor in cultured cerebellar granule cells

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Identifiers

DOI

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13 February 2018

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13 February 2018