(Q48640006)

13 February 2018

title

Aberrant expression of bcl-2 gene family in Down's syndrome brains (English)

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13 February 2018

author

Fumitaka Oyama

2

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13 February 2018

author name string

Sawa A

1

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13 February 2018

Cairns NJ

3

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13 February 2018

Amano N

4

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13 February 2018

Matsushita M

5

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13 February 2018

publication date

1 August 1997

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13 February 2018

published in

Molecular Brain Research

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13 February 2018

volume

48

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13 February 2018

issue

1

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13 February 2018

page(s)

53-59

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13 February 2018

Expression of the bcl-2 gene family in normal and malignant breast tissue: low bax-alpha expression in tumor cells correlates with resistance towards apoptosis

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Overexpression of BCL-2 in transgenic mice protects neurons from naturally occurring cell death and experimental ischemia

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Bcl-2 heterodimerizes in vivo with a conserved homolog, Bax, that accelerates programmed cell death

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Immunoreactivity for Bcl-2 protein within neurons in the Alzheimer's disease brain increases with disease severity

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Multiple Bcl-2 family members demonstrate selective dimerizations with Bax

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7 January 2021

Immunohistochemical evidence for apoptosis in Alzheimer's disease.

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7 January 2021

Up-regulation of Bcl-2 is associated with neuronal DNA damage in Alzheimer's disease.

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7 January 2021

Protease inhibitor domain encoded by an amyloid protein precursor mRNA associated with Alzheimer's disease

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7 January 2021

Expression of members of the bcl-2 gene family in the immature rat ovary: equine chorionic gonadotropin-mediated inhibition of granulosa cell apoptosis is associated with decreased bax and constitutive bcl-2 and bcl-xlong messenger ribonucleic acid

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7 January 2021

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Analysis of the structure, transcripts, and protein products of bcl-2, the gene involved in human follicular lymphoma

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7 January 2021

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7 January 2021

Bad, a heterodimeric partner for Bcl-XL and Bcl-2, displaces Bax and promotes cell death

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7 January 2021

Neurotoxicity of a fragment of the amyloid precursor associated with Alzheimer's disease

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7 January 2021

BH1 and BH2 domains of Bcl-2 are required for inhibition of apoptosis and heterodimerization with Bax.

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7 January 2021

Degeneration in vitro of post-mitotic neurons overexpressing the Alzheimer amyloid protein precursor

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7 January 2021

10.1016/S0169-328X(97)00078-8

Identifiers

DOI

1 reference

13 February 2018

1 reference

13 February 2018