(Q48643474)

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Altered activity of the inhibitory guanyl nucleotide-binding component (Ni) induced by pertussis toxin. Uncoupling of Ni from receptor with continued coupling of Ni to the catalytic unit

scientific article published on July 25, 1984

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26 October 2019

Altered activity of the inhibitory guanyl nucleotide-binding component (Ni) induced by pertussis toxin. Uncoupling of Ni from receptor with continued coupling of Ni to the catalytic unit (English)

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26 October 2019

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author name string

T. E. Cote

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E. A. Frey

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R. D. Sekura

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1 July 1984

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26 October 2019

25 July 1984

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6

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full work available at URL

http://intl.jbc.org/cgi/content/abstract/259/14/8693

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Journal of Biological Chemistry

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26 October 2019

259

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26 October 2019

8693-8698

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Effect of in Vivo Pretreatment of Rats with a New Protein Purified fromBordetellapertussis onin VitroSecretion of Insulin: Role of Calcium

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Pertussis toxin blocks somatostatin's inhibition of stimulated cyclic AMP accumulation in anterior pituitary tumor cells

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American Journal of Physiology

8

244

E499

author name string

Cronin

publication date

1983

1 reference

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Pertussis toxin blocks the somatostatin-induced inhibition of growth hormone release and adenosine 3',5'-monophosphate accumulation

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Direct modification of the membrane adenylate cyclase system by islet-activating protein due to ADP-ribosylation of a membrane protein

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Stimulation and inhibition of adenylyl cyclases mediated by distinct regulatory proteins

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Pertussis toxin substrate, the putative Ni component of adenylyl cyclases, is an alpha beta heterodimer regulated by guanine nucleotide and magnesium

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D-2 dopamine receptor-mediated inhibition of adenylate cyclase activity in the intermediate lobe of the rat pituitary gland requires guanosine 5'-triphosphate.

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Guanyl nucleotides participate in the beta-adrenergic stimulation of adenylate cyclase activity in the intermediate lobe of the rat pituitary gland.

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The dopamine receptor in the intermediate lobe of the rat pituitary gland is negatively coupled to adenylate cyclase

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Selective decrease in the affinity of D2 dopamine receptor for agonist induced by islet-activating protein, pertussis toxin, associated with ADP-ribosylation of the specific membrane protein of bovine striatum

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Guanine nucleotide activation and inhibition of adenylate cyclase as modified by islet-activating protein, pertussis toxin, in mouse 3T3 fibroblasts

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publication date

1982

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Europe PubMed Central

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26 October 2019

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