(Q48670206)

14 February 2018

title

Diaphragm and cardiac mitochondrial creatine kinases are impaired in sepsis. (English)

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14 February 2018

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Leigh A Callahan

1

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14 February 2018

Gerald S Supinski

series ordinal

2

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14 February 2018

language of work or name

English

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publication date

17 August 2006

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14 February 2018

Journal of Applied Physiology

published in

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14 February 2018

volume

102

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14 February 2018

issue

1

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14 February 2018

page(s)

44-53

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14 February 2018

A mitochondrial perspective on cell death

cites work

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Sepsis induces diaphragm electron transport chain dysfunction and protein depletion

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Actions of the creatine analogue beta-guanidinopropionic acid on rat heart mitochondria

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Ileal VO(2)-O(2) alterations induced by endotoxin correlate with severity of mitochondrial injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Bench-to-bedside review: Cytopathic hypoxia

21 January 2018

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https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Cardiac troponins and creatine kinase content of striated muscle in common laboratory animals

21 January 2018

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https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Importance of mitochondrial transmembrane processes in human mitochondriopathies

21 January 2018

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https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Microcirculatory oxygenation and shunting in sepsis and shock

21 January 2018

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https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Apoptotic cell death in the mouse retinal ganglion cell layer is induced in vivo by the excitatory amino acid homocysteine

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Septic shock in humans. Advances in the understanding of pathogenesis, cardiovascular dysfunction, and therapy

21 January 2018

1 reference

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21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Mitochondrial dysfunction in sepsis

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Octameric mitochondrial creatine kinase induces and stabilizes contact sites between the inner and outer membrane

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Mitochondrial creatine kinase is critically necessary for normal myocardial high-energy phosphate metabolism

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Mitochondrial creatine kinase is a prime target of peroxynitrite-induced modification and inactivation.

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Oxidative metabolism in sepsis and sepsis syndrome

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Different sensitivity of rabbit heart and skeletal muscle to endotoxin-induced impairment of mitochondrial function

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Reduced creatine kinase activity in transgenic amyotrophic lateral sclerosis mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1152%2FJAPPLPHYSIOL.01204.2005

Differential effects of peroxynitrite on human mitochondrial creatine kinase isoenzymes. Inactivation, octamer destabilization, and identification of involved residues

21 January 2018

1 reference

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10.1152/JAPPLPHYSIOL.01204.2005

21 January 2018

Identifiers

DOI

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14 February 2018

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14 February 2018