(Q48697372)

14 February 2018

title

8-OH-DPAT and MK-801 affect epileptic activity independently of vigilance. (English)

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14 February 2018

author

György Bagdy

4

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14 February 2018

author name string

Filakovszky J

1

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14 February 2018

Kantor S

2

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14 February 2018

Halasz P

3

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14 February 2018

publication date

1 June 2001

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14 February 2018

Neurochemistry International

published in

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14 February 2018

volume

38

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14 February 2018

issue

7

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14 February 2018

page(s)

551-556

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14 February 2018

Calcium-dependent regulation of genetically determined spike and waves by the reticular thalamic nucleus of rats.

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Comparison of relative potencies of i.v. and i.c.v. administered 8-OH-DPAT gives evidence of different sites of action for hypothermia, lower lip retraction and tail flicks.

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Thalamic NMDA receptors in the gamma-hydroxybutyrate model of absence seizures: a cerebral microinjection study in rats

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Evidence that activation of GABA receptors in the substantia nigra suppresses spontaneous spike-and-wave discharges in the rat.

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Spike-wave discharges and sleep-wake states in rats with absence epilepsy.

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A serotonin-1A receptor agonist and an N-methyl-D-aspartate receptor antagonist oppose each others effects in a genetic rat epilepsy model.

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The 5-HT1A agonist 8-OH-DPAT increases the number of spike-wave discharges in a genetic rat model of absence epilepsy.

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Relationship between spike-wave discharges and vigilance levels in rats with spontaneous petit mal-like epilepsy.

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Evaluation of the 5-hydroxytryptamine receptor agonist 8-hydroxy-2-(di-n-propylamino)tetralin in different rodent models of epilepsy

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The behavioural effects of MK-801 in rats: involvement of dopaminergic, serotonergic and noradrenergic systems

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The receptor antagonist MK-801 induces increases in dopamine and serotonin metabolism in several brain regions of rats

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Basic mechanisms of generalized absence seizures

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Abnormalities in brain serotonin concentration, high-affinity uptake, and tryptophan hydroxylase activity in severe-seizure genetically epilepsy-prone rats.

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7 January 2021

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Identifiers

DOI

1 reference

14 February 2018

1 reference

14 February 2018