(Q48763738)

15 February 2018

title

Role of nitric oxide in the cerebellar degeneration during methylmercury intoxication. (English)

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15 February 2018

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T Yamashita

1

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15 February 2018

Y Ando

2

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15 February 2018

N Sakashita

3

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15 February 2018

K Hirayama

4

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15 February 2018

Y Tanaka

5

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15 February 2018

K Tashima

6

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15 February 2018

M Uchino

7

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15 February 2018

M Ando

8

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15 February 2018

publication date

1 March 1997

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15 February 2018

Biochimica et Biophysica Acta

published in

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15 February 2018

volume

1334

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15 February 2018

issue

2-3

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15 February 2018

page(s)

303-311

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15 February 2018

Nitric oxide release accounts for the biological activity of endothelium-derived relaxing factor

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Relative sparing of nitric oxide synthase–containing neurons in the hippocampal formation in Alzheimer's disease

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Localization of nitric oxide synthase indicating a neural role for nitric oxide

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Focal cerebellar and cerebellar atrophy in a human subject due to organic mercury compounds

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Neuronal NADPH diaphorase is a nitric oxide synthase

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Nitric oxide mediates glutamate-linked enhancement of cGMP levels in the cerebellum

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NMDA receptor activation induces nitric oxide synthesis from arginine in rat brain slices

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Methylmercury-induced alterations in excitatory amino acid transport in rat primary astrocyte cultures.

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Two classes of N-methyl-D-aspartate recognition sites: differential distribution and differential regulation by glycine

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Nitric oxide: a cytotoxic activated macrophage effector molecule

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Chromosomal assignment of the second locus for autosomal dominant cerebellar ataxia (SCA2) to chromosome 12q23-24.1

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Olivopontocerebellar degeneration. Clinical and ultrastructural abnormalities.

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Dominant spinopontine atrophy. Report of two additional members of family W

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Identifiers

DOI

1 reference

15 February 2018

1 reference

15 February 2018