(Q48770511)

English

Pretreatment with neurokinin substance P but not with cholecystokinin-8S can alleviate functional deficits of partial nigrostriatal 6-hydroxydopamine lesion.

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Pretreatment with neurokinin substance P but not with cholecystokinin-8S can alleviate functional deficits of partial nigrostriatal 6-hydroxydopamine lesion. (English)

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

0 references

cholecystokinin

1 reference

based on heuristic

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author name string

Nikolaus S

1

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Huston JP

2

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Körber B

3

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Thiel C

4

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Schwarting RK

5

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

publication date

1 January 1997

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

18

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

8

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

1161-1168

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

Cholecystokinin

1 reference

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Efficacy of brain-derived neurotrophic factor and neurotrophin-3 on neurochemical and behavioral deficits associated with partial nigrostriatal dopamine lesions

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Saturable transport of peptides across the blood-brain barrier

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Neurokinin-3 receptors modulate dopamine cell function and alter the effects of 6-hydroxydopamine

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Functions of brain-derived neurotrophic factor, insulin-like growth factor-I and basic fibroblast growth factor in the development and maintenance of dopaminergic neurons

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Effects of substance P on extracellular dopamine in neostriatum and nucleus accumbens.

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Substance P decreases extracellular concentrations of acetylcholine in neostriatum and nucleus accumbens in vivo: possible relevance for the central processing of reward and aversion.

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Relationship of changes in spontaneous motor activity to spontaneous circling in rats with unilateral 6-hydroxydopamine lesions of the substantia nigra

1 reference

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Substance P-dopamine relationship in the rat substantia nigra: a light and electron microscopy study of double immunocytochemically labeled materials

1 reference

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Cholecystokinin-dopamine interactions

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Substance P in the substantia nigra

1 reference

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Immunoreactive substance P in the substantia nigra of the monkey: light and electron microscopic localization

1 reference

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Behavioral asymmetries and recovery in rats with different degrees of unilateral striatal dopamine depletion

1 reference

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Ceruletide suppresses rotational behavior in lesioned rats via CCKA receptors

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Substance P enhancement of inhibitory avoidance learning: mediation by the N-terminal sequence.

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Evidence for mnemotropic action of cholecystokinin fragments Boc-CCK-4 and CCK-8S.

1 reference

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Chronic administration of neurokinin SP improves maze performance in aged Rattus norvegicus

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Neurotrophic factor therapy for nervous system degenerative diseases

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The relationship between loss of dopamine nerve terminals, striatal [3H]spiroperidol binding and rotational behavior in unilaterally 6-hydroxydopamine-lesioned rats

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A subpopulation of mesencephalic dopamine neurons projecting to limbic areas contains a cholecystokinin-like peptide: evidence from immunohistochemistry combined with retrograde tracing

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The basal ganglia-orofacial system: studies on neurobehavioral plasticity and sensory-motor tuning

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Sequence-specific effects of neurokinin substance P on memory, reinforcement, and brain dopamine activity

1 reference

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The role of neuropeptides in learning: focus on the neurokinin substance P.

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Trophic effect of various neuropeptides on the cultured ventral spinal cord of rat embryo

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Substance P Counteracts Neurotoxin Damage on Norepinephrine Neurons in Rat Brain During Ontogeny

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Substance P modifies the 6-hydroxydopamine induced alteration of postnatal development of central noradrenaline neurons

1 reference

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Protective effect of CCK-8 and ceruletide on glutamate-induced neuronal cell death in rat neuron cultures: Possible involvement of CCK-B receptors

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An in vivo model for the neurodegenerative effects of beta amyloid and protection by substance P

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Substance P in the human brain

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Substance P receptor binding sites are expressed by glia in vivo after neuronal injury

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Effect of cholecystokinin-octapeptide on dopamine release from slices of cat caudate nucleus

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Recovery from unilateral 6-hydroxydopamine lesion of substantia nigra promoted by the neurotachykinin substance P 1-11.

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Effects of reducing sensory-motor feedback on the appearance of crossed nigro-thalamic projections and recovery from turning induced by unilateral substantia nigra lesions.

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Behavioral analysis of asymmetries induced by unilateral 6-OHDA injections into the substantia nigra.

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Accelerated regeneration of central catecholamine fibers in cat occipital cortex: effects of substance P

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Stimulatory effects of substance P on neurite extension and cyclic AMP levels in cultured neuroblastoma cells

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Changes in tachykinin and actin gene expression in rat sensory neurones after cutting or crushing the sciatic nerve

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Effect of cutting or crushing the rat sciatic nerve on synthesis of substance P by isolated L5 dorsal root ganglia

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Reinforcing effects of peripherally administered substance P and its C-terminal sequence pGlu6-SP6-11 in the rat.

1 reference

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Striatonigral GABA, dynorphin, substance P and neurokinin A modulation of nigrostriatal dopamine release: evidence for direct regulatory mechanisms

1 reference

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Effects of cholecystokinin peptides and neurotensin on dopamine release and metabolism in the rostral and caudal part of the nucleus accumbens using intracerebral dialysis in the anaesthetized rat

1 reference

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Short-term effects of ether, equithesin and droperidol/fentanyl on catecholamine and indolamine metabolism in the brain of the rat.

1 reference

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Unilateral 6-hydroxydopamine lesions of meso-striatal dopamine neurons and their physiological sequelae

1 reference

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Relationships between indices of behavioral asymmetries and neurochemical changes following mesencephalic 6-hydroxydopamine injections.

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Synaptic connections of substance P-immunoreactive nerve terminals in the substantia nigra of the rat. A correlated light- and electron-microscopic study.

1 reference

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Effects of chronic substance P treatment and intracranial fetal grafts on learning after hippocampal kainic acid lesions.

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7 January 2021

based on heuristic

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Facilitation of conditioned inhibitory avoidance by post-trial peripheral injection of substance P.

1 reference

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7 January 2021

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1 reference

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7 January 2021

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Neurotrophic effects of substance P on hippocampal neurons in vitro

1 reference

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NGF rescues substance P expression but not neurofilament or tubulin gene expression in axotomized sensory neurons

1 reference

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Neurotrophic and neurotoxic effects of amyloid beta protein: reversal by tachykinin neuropeptides

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Identifiers

10.1016/S0196-9781(97)00181-2

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

15 February 2018

http://europepmc.org/abstract/MED/9396056

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