(Q48777649)

15 February 2018

title

Upregulation of transcription factors controlling MHC expression in multiple sclerosis lesions. (English)

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15 February 2018

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Gobin SJ

1

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Montagne L

2

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Van Zutphen M

3

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Van Der Valk P

4

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Van Den Elsen PJ

5

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De Groot CJ

6

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publication date

1 October 2001

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15 February 2018

published in

Glia

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15 February 2018

volume

36

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issue

1

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page(s)

68-77

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Role of macrophages/microglia in multiple sclerosis and experimental allergic encephalomyelitis

cites work

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Detection of MHC class II-antigens on macrophages and microglia, but not on astrocytes and endothelia in active multiple sclerosis lesions

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Activation of NF-kappaB and c-jun transcription factors in multiple sclerosis lesions. Implications for oligodendrocyte pathology.

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Regulation of MHC class II expression in the central nervous system

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Expression of transforming growth factor (TGF)-beta1, -beta2, and -beta3 isoforms and TGF-beta type I and type II receptors in multiple sclerosis lesions and human adult astrocyte cultures

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The bare lymphocyte syndrome: molecular clues to the transcriptional regulation of major histocompatibility complex class II genes

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Microglia: Intrinsic immuneffector cell of the brain

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The RFX complex is crucial for the constitutive and CIITA-mediated transactivation of MHC class I and beta2-microglobulin genes

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Microglia as mediators of inflammatory and degenerative diseases

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Transcription factor NF-kappaB and inhibitor I kappaBalpha are localized in macrophages in active multiple sclerosis lesions.

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Tumor necrosis factor identified in multiple sclerosis brain

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Differential selectivity of CIITA promoter activation by IFN-gamma and IRF-1 in astrocytes and macrophages: CIITA promoter activation is not affected by TNF-alpha

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Absence of neurological deficits following extensive demyelination in a class I-deficient murine model of multiple sclerosis

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Identification of lymphotoxin and tumor necrosis factor in multiple sclerosis lesions

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Expression of specific chemokines and chemokine receptors in the central nervous system of multiple sclerosis patients

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Indictment of the microglia as the villain in multiple sclerosis

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Regulation of MHC gene expression

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Shared regulatory elements in the promoters of MHC class I and class II genes

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Molecular mechanisms of microglial activation

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21 January 2018

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Site alpha is crucial for two routes of IFN gamma-induced MHC class I transactivation: the ISRE-mediated route and a novel pathway involving CIITA

21 January 2018

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Astrocytic factors deactivate antigen presenting cells that invade the central nervous system.

21 January 2018

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Microglia-derived macrophages in early multiple sclerosis plaques

21 January 2018

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Activation of the MHC class II transactivator CIITA by interferon-gamma requires cooperative interaction between Stat1 and USF-1

21 January 2018

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Is there a balance between microglia and astrocytes in regulating Th1/Th2-cell responses and neuropathologies?

21 January 2018

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Identifiers

DOI

10.1002/GLIA.1096

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15 February 2018

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15 February 2018