(Q48823958)

15 February 2018

title

Cholinergic activation of phosphoinositide signaling is impaired in Alzheimer's disease brain. (English)

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15 February 2018

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R S Jope

1

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15 February 2018

L Song

series ordinal

2

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15 February 2018

R E Powers

series ordinal

3

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15 February 2018

publication date

1 January 1997

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15 February 2018

published in

Neurobiology of Aging

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15 February 2018

volume

18

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15 February 2018

page(s)

111-120

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15 February 2018

issue

1

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15 February 2018

A neurochemical approach for studying response to acetylcholine in Alzheimer's disease

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Cholinergic and serotonergic stimulation of phosphoinositide hydrolysis is decreased in Alzheimer's disease.

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Amyloid beta protein disruption of cholinergic and growth factor phospholipase C signals could underlie cognitive and neurodegerative aspects of Alzheimer's disease

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Diminished muscarinic receptor-stimulated [3H]-PIP2 hydrolysis in Alzheimer's disease

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Inositol Lipids and Signal Transduction in the Nervous System: An Update

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Loss of high-affinity agonist binding to M1 muscarinic receptors in Alzheimer's disease: implications for the failure of cholinergic replacement therapies.

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Phosphoinositide hydrolysis, G alpha q, phospholipase C, and protein kinase C in post mortem human brain: effects of post mortem interval, subject age, and Alzheimer's disease.

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Impaired phosphoinositide hydrolysis in Alzheimer's disease brain.

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[3H]PtdIns hydrolysis in postmortem human brain membranes is mediated by the G-proteins Gq/11 and phospholipase C-beta

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Transmembrane signaling through phospholipase C in human cortical membranes.

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Receptor-mediated activation of G proteins is reduced in postmortem brains from Alzheimer's disease patients.

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7 January 2021

Coupling of muscarinic receptors to GTP proteins in postmortem human brain--alterations in Alzheimer's disease.

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Identifiers

DOI

1 reference

15 February 2018

PubMed ID

8983039

1 reference

15 February 2018