(Q48847222)

English

Intracellular chelation of calcium prevents cell damage following severe hypoxia in the rat cerebral cortex as studied by NMR spectroscopy ex vivo.

scientific article published in December 1996

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scholarly article

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

Intracellular chelation of calcium prevents cell damage following severe hypoxia in the rat cerebral cortex as studied by NMR spectroscopy ex vivo. (English)

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

author name string

O Gröhn

1

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

R Kauppinen

2

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

publication date

1 December 1996

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

20

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

6

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

509-514

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

describes a project that uses

NMR spectroscopy

1 reference

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Calcium-mediated neurotoxicity: relationship to specific channel types and role in ischemic damage

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Source specificity of early calcium neurotoxicity in cultured embryonic spinal neurons

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Properties of neuroprotective cell-permeant Ca2+ chelators: effects on [Ca2+]i and glutamate neurotoxicity in vitro.

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Calcium-mediated damage following hypoxia in cerebral cortex ex vivo studied by NMR spectroscopy. Evidence for direct involvement of voltage-gated Ca(2+)-channels.

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Involvement of intracellular stores in the Ca2+ responses to N-Methyl-D-aspartate and depolarization in cerebellar granule cells.

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Use of 1,2-bis(2-amino-5-fluorophenoxy)ethane-N,N,N',N'-tetraacetic acid (5FBAPTA) in the measurement of free intracellular calcium in the brain by 19F-nuclear magnetic resonance spectroscopy.

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Bioenergetic recovery following ischemia in brain slices studied by 31P-NMR spectroscopy: differential age effect of depolarization mediated by endogenous nitric oxide.

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Neither moderate hypoxia nor mild hypoglycaemia alone causes any significant increase in cerebral [Ca2+]i: only a combination of the two insults has this effect. A 31P and 19F NMR study

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Neuroprotection in vitro and in vivo by cell membrane-permeant Ca2+ chelators

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Acute cerebral ischaemia: concurrent changes in cerebral blood flow, energy metabolites, pH, and lactate measured with hydrogen clearance and 31P and 1H nuclear magnetic resonance spectroscopy. III. Changes following ischaemia

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Delayed ("secondary") cerebral energy failure after acute hypoxia-ischemia in the newborn piglet: continuous 48-hour studies by phosphorus magnetic resonance spectroscopy

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Increased apoptosis in the cingulate sulcus of newborn piglets following transient hypoxia-ischaemia is related to the degree of high energy phosphate depletion during the insult

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1H nuclear magnetic resonance spectroscopy identifies neural cell types: a promising step for neuroimaging?

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Changes in N-acetyl-aspartate content during focal and global brain ischemia of the rat

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Identifiers

10.1016/S0143-4160(96)90093-7

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

1 reference

Europe PubMed Central

16 February 2018

http://europepmc.org/abstract/MED/8985596

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