(Q49021460)

English

Tumor necrosis factor-alpha neutralization reduced cerebral edema through inhibition of matrix metalloproteinase production after transient focal cerebral ischemia.

scientific article published in August 2005

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Tumor necrosis factor-alpha neutralization reduced cerebral edema through inhibition of matrix metalloproteinase production after transient focal cerebral ischemia. (English)

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

cerebral infarction

1 reference

based on heuristic

inferred from title

1 reference

based on heuristic

inferred from title

1

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

author name string

Camelia R Ban

2

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Takayuki Naya

3

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Tsutomu Takahashi

4

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Peng Guo

5

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Xiao-yu R Song

6

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Masakazu Kohno

7

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

publication date

1 August 2005

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Journal of Cerebral Blood Flow & Metabolism

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

25

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

8

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

959-967

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

Tumor necrosis factor-alpha. A mediator of focal ischemic brain injury.

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Prolonged activation of jun and collagenase genes by tumour necrosis factor-alpha

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Altered neuronal and microglial responses to excitotoxic and ischemic brain injury in mice lacking TNF receptors

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Activation systems for latent matrix metalloproteinase-2 are upregulated immediately after focal cerebral ischemia.

1 reference

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Mitogen-activated protein kinases mediate matrix metalloproteinase-9 expression in vascular smooth muscle cells

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Potential of anticytokine therapies in central nervous system ischaemia

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Inhibition of tumor necrosis factor-alpha reduces focal cerebral ischemic injury in the spontaneously hypertensive rat.

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Anti-TNF-alpha antibodies suppress the development of experimental autoimmune myasthenia gravis

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Development of anti-TNF therapy for rheumatoid arthritis

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Disruption of epithelial cell-matrix interactions induces apoptosis

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Cytokine-stimulated human vascular smooth muscle cells synthesize a complement of enzymes required for extracellular matrix digestion

1 reference

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Ischemic and excitotoxic brain injury is enhanced in mice lacking the p55 tumor necrosis factor receptor.

1 reference

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Cellular localization of tumor necrosis factor alpha following focal cerebral ischemia in mice

1 reference

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Microglia and macrophages are the major source of tumor necrosis factor in permanent middle cerebral artery occlusion in mice

1 reference

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Microvascular basal lamina antigens disappear during cerebral ischemia and reperfusion

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Regulation of matrix metalloproteinase expression in human vein and microvascular endothelial cells. Effects of tumour necrosis factor alpha, interleukin 1 and phorbol ester

1 reference

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Duration threshold of induced hypertension on cerebral blood flow, energy metabolism, and edema after transient forebrain ischemia in gerbils.

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TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

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Production of MMPs in human cerebral endothelial cells and their role in shedding adhesion molecules

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Morbillivirus infection of the mouse central nervous system induces region-specific upregulation of MMPs and TIMPs correlated to inflammatory cytokine expression

1 reference

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Effect of brain edema on infarct volume in a focal cerebral ischemia model in rats.

1 reference

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Hypoxic preconditioning protects cultured neurons against hypoxic stress via TNF-alpha and ceramide.

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Tumor necrosis factor-alpha expression in ischemic neurons

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Experimental cerebral infarctioneffects of pentobarbital in Mongolian gerbils.

1 reference

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7 January 2021

based on heuristic

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Inhibition of tumor necrosis factor and amelioration of brain infarction in mice

1 reference

https://api.crossref.org/works/10.1038%2FSJ.JCBFM.9600086

7 January 2021

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TNF-alpha pretreatment induces protective effects against focal cerebral ischemia in mice.

1 reference

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Pathophysiological process after transient ischemia of the middle cerebral artery in the rat.

1 reference

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Elevated expression of membrane type 1 metalloproteinase (MT1-MMP) in reactive astrocytes following neurodegeneration in mouse central nervous system

1 reference

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Inhibition of anchorage-dependent cell spreading triggers apoptosis in cultured human endothelial cells

1 reference

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Protein extravasation and cellular uptake after high-dose human-albumin treatment of transient focal cerebral ischemia in rats.

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Tumor necrosis factor-alpha-induced gelatinase B causes delayed opening of the blood-brain barrier: an expanded therapeutic window

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Proteolytic cascade enzymes increase in focal cerebral ischemia in rat

1 reference

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Metalloproteinase inhibition blocks edema in intracerebral hemorrhage in the rat.

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Immunohistochemistry of matrix metalloproteinases in reperfusion injury to rat brain: activation of MMP-9 linked to stromelysin-1 and microglia in cell cultures.

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Evolution of cerebral tumor necrosis factor-alpha production during human ischemic stroke

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Tumor necrosis factor alpha induces a metalloprotease-disintegrin, ADAM8 (CD 156): implications for neuron-glia interactions during neurodegeneration

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RETRACTED: A new mechanism of neurodegeneration: a proinflammatory cytokine inhibits receptor signaling by a survival peptide

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Tumor necrosis factor alpha expression produces increased blood-brain barrier permeability following temporary focal cerebral ischemia in mice.

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Early TNF-alpha levels correlate with ischaemic stroke severity.

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Reversible middle cerebral artery occlusion without craniectomy in rats

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Identifiers

10.1038/SJ.JCBFM.9600086

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

17 February 2018

http://europepmc.org/abstract/MED/15729288

ResearchGate publication ID

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