(Q49023123)

English

NF-kappaB is involved in the survival of cerebellar granule neurons: association of IkappaBbeta [correction of Ikappabeta] phosphorylation with cell survival.

scientific article published in February 2001

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scholarly article

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

NF-kappaB is involved in the survival of cerebellar granule neurons: association of IkappaBbeta [correction of Ikappabeta] phosphorylation with cell survival. (English)

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

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phosphorylation

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author name string

E Koulich

1

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

T Nguyen

2

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

K Johnson

3

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

C Giardina

4

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

S D'mello

5

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

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publication date

1 February 2001

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Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

number of pages

11

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Journal of Neurochemistry

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Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

76

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Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

4

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Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

1188-1198

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

L-929 cells harboring ectopically expressed RelA resist curcumin-induced apoptosis

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Tumor necrosis factors alpha and beta protect neurons against amyloid beta-peptide toxicity: evidence for involvement of a kappa B-binding factor and attenuation of peroxide and Ca2+ accumulation.

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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DNA-dependent protein kinase is one of a subset of autoantigens specifically cleaved early during apoptosis

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Inhibition of the RelA(p65) NF-kappaB subunit by Egr-1.

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Suppression of tumor necrosis factor-induced cell death by inhibitor of apoptosis c-IAP2 is under NF-kappaB control

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Global cerebral ischemia activates nuclear factor-kappa B prior to evidence of DNA fragmentation.

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Induction of apoptosis in cerebellar granule neurons by low potassium: inhibition of death by insulin-like growth factor I and cAMP.

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Regulation of neuronal survival by the serine-threonine protein kinase Akt

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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NF-kappa B and Rel proteins: evolutionarily conserved mediators of immune responses

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Nuclear factor-kappaB mediates the cell survival-promoting action of activity-dependent neurotrophic factor peptide-9

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Neuroprotection by aspirin and sodium salicylate through blockade of NF-kappaB activation.

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Rapid proteolysis of I kappa B-alpha is necessary for activation of transcription factor NF-kappa B

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Nuclear translocation of NF-kappaB is increased in dopaminergic neurons of patients with parkinson disease

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Potential involvement of the transcription factor NF-kappa B in neurological disorders

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Transcription factor NF-kappaB is activated in primary neurons by amyloid beta peptides and in neurons surrounding early plaques from patients with Alzheimer disease

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Inhibition of NF-kappaB potentiates amyloid beta-mediated neuronal apoptosis

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Phosphorylation meets ubiquitination: the control of NF-[kappa]B activity

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Apoptosis overrides survival signals through a caspase-mediated dominant-negative NF-kappa B loop

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Suppression of steady-state, but not stimulus-induced NF-kappaB activity inhibits alphavirus-induced apoptosis

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Inhibition of nuclear translocation of transcription factor NF-kappa B by a synthetic peptide containing a cell membrane-permeable motif and nuclear localization sequence

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21 January 2018

DNA-dependent protein kinase phosphorylation of IkappaB alpha and IkappaB beta regulates NF-kappaB DNA binding properties

1 reference

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Blockade of T-cell activation by dithiocarbamates involves novel mechanisms of inhibition of nuclear factor of activated T cells.

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Activation of NF-kappaB protects hippocampal neurons against oxidative stress-induced apoptosis: evidence for induction of manganese superoxide dismutase and suppression of peroxynitrite production and protein tyrosine nitration.

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21 January 2018

Roles of nuclear factor kappaB in neuronal survival and plasticity

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Inactivation of IkappaBbeta by the tax protein of human T-cell leukemia virus type 1: a potential mechanism for constitutive induction of NF-kappaB

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Recruitment of CBP/p300 by the IFN beta enhanceosome is required for synergistic activation of transcription

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Cytokine-induced nuclear factor kappa B activation promotes the survival of developing neurons.

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Inhibition of phosphatidylinositol 3-kinase activity blocks depolarization- and insulin-like growth factor I-mediated survival of cerebellar granule cells.

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Caspase-3 expression by cerebellar granule neurons is regulated by calcium and cyclic AMP.

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Regulation of NF-kappaB by cyclin-dependent kinases associated with the p300 coactivator

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Regulation of IkappaB beta in WEHI 231 mature B cells

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Nuclear factor-kappa B in rat brain: enhanced DNA-binding activity following convulsant-induced seizures.

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Dithiocarbamates as potent inhibitors of nuclear factor kappa B activation in intact cells

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Inhibition of nuclear factor-kappaB-mediated transcription by association with the amino-terminal enhancer of split, a Groucho-related protein lacking WD40 repeats

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Activation of nuclear factor-kappaB-dependent transcription by tumor necrosis factor-alpha is mediated through phosphorylation of RelA/p65 on serine 529.

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NF-kappaB induces expression of the Bcl-2 homologue A1/Bfl-1 to preferentially suppress chemotherapy-induced apoptosis

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NF-kappaB subunit p65 binds to 53BP2 and inhibits cell death induced by 53BP2

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Phosphorylation of NF-kappa B p65 by PKA stimulates transcriptional activity by promoting a novel bivalent interaction with the coactivator CBP/p300

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Functional Interplay Between Nuclear Factor-κB and c-Jun Integrated by Coactivator p300 Determines the Survival of Nerve Growth Factor-Dependent PC12 Cells

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Identifiers

10.1046/J.1471-4159.2001.00134.X

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

1 reference

Europe PubMed Central

17 February 2018

http://europepmc.org/abstract/MED/11181838

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