(Q49044104)

English

Blockade of GABA(A) receptors in the paraventricular nucleus of the hypothalamus attenuates voluntary ethanol intake and activates the hypothalamic-pituitary-adrenocortical axis.

scientific article published on 18 July 2011

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scholarly article

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Blockade of GABA(A) receptors in the paraventricular nucleus of the hypothalamus attenuates voluntary ethanol intake and activates the hypothalamic-pituitary-adrenocortical axis. (English)

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

author name string

Jing Li

1

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Weiliang Bian

2

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Vaidehi Dave

3

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Jiang-Hong Ye

4

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

publication date

18 July 2011

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Addiction Biology

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

16

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

4

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

600-614

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

Suppression of the HPA axis stress-response: implications for relapse

1 reference

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Disturbances of hypothalamic-pituitary-adrenal axis functioning during ethanol withdrawal in six men.

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Increased Salivary Cortisol Concentrations During Chronic Alcohol Intoxication in a Naturalistic Clinical Sample of Men

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GABAergic control of neuropeptide gene expression in parvocellular neurons of the hypothalamic paraventricular nucleus

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Opioids in the hypothalamic paraventricular nucleus stimulate ethanol intake

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Local gamma-aminobutyric acid and glutamate circuit control of hypophyseotrophic corticotropin-releasing factor neuron activity in the paraventricular nucleus of the hypothalamus

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Functional role of local GABAergic influences on the HPA axis

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GABA: a dominant neurotransmitter in the hypothalamus.

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Neuropeptide Y levels in ethanol-naive alcohol-preferring and nonpreferring rats and in Wistar rats after ethanol exposure

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Chronic ethanol exposure leads to divergent control of dopaminergic synapses in distinct target regions

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Altered chloride homeostasis removes synaptic inhibitory constraint of the stress axis

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c-Fos and related immediate early gene products as markers of activity in neuroendocrine systems

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A GABA-neuropeptide Y (NPY) interplay in LH release

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Acute and delayed effects of picrotoxin on the adrenocorticotropic system of rats

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Interacting appetite-regulating pathways in the hypothalamic regulation of body weight

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Salivary cortisol measurements during a medically assisted alcohol withdrawal.

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GABA and hypothalamic feeding systems. I. Topographic analysis of the effects of microinjections of muscimol

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Neuroendocrine pathways of addictive behaviour

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GABAergic mechanisms constraining the activity of the hypothalamo-pituitary-adrenocortical axis

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Time course of vasopressin and oxytocin secretion after stress in adrenalectomized rats.

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Prolonged exposure to intermittent alcohol vapors blunts hypothalamic responsiveness to immune and non-immune signals

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21 January 2018

Region-specific induction of FosB/ΔFosB by voluntary alcohol intake: effects of naltrexone

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Mechanisms of reversible GABAA receptor plasticity after ethanol intoxication.

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Blunted stress cortisol response in abstinent alcoholic and polysubstance-abusing men.

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GABAergic mediation of stress-induced secretion of corticosterone and oxytocin, but not prolactin, by the hypothalamic paraventricular nucleus

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Adaptation in patterns of c-fos expression in the brain associated with exposure to either single or repeated social stress in male rats.

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GABA regulates the rat hypothalamic-pituitary-adrenocortical axis via different GABA-A receptor alpha-subtypes

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GABAergic innervation of corticotropin-releasing hormone (CRH)-secreting parvocellular neurons and its plasticity as demonstrated by quantitative immunoelectron microscopy

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Locomotor activity initiated by microinfusions of picrotoxin into the ventral tegmental area

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Blocking GABA(A) receptors in the anterior ventral tegmental area attenuates ethanol intake of the alcohol-preferring P rat.

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Chronic daily ethanol and withdrawal: 1. Long-term changes in the hypothalamo-pituitary-adrenal axis.

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Alpha4-containing GABAA receptors in the nucleus accumbens mediate moderate intake of alcohol

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Alcohol self-administration acutely stimulates the hypothalamic-pituitary-adrenal axis, but alcohol dependence leads to a dampened neuroendocrine state

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Local origins of some GABAergic projections to the paraventricular and supraoptic nuclei of the hypothalamus in the rat.

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Decreased brain reward produced by ethanol withdrawal

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Stress-induced expression of immediate early genes in the brain and peripheral organs of the rat.

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Prolonged alcohol intake leads to reversible depression of corticotropin-releasing hormone and vasopressin immunoreactivity and mRNA levels in the parvocellular neurons of the paraventricular nucleus

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Identifiers

10.1111/J.1369-1600.2011.00344.X

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/21762292

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