(Q49061721)

18 February 2018

title

Reproducible peracute glutamate-induced focal lesions of the normal rat brain using microdialysis (English)

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18 February 2018

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Landolt H

1

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18 February 2018

Fujisawa H

2

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18 February 2018

Graham DI

3

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18 February 2018

Maxwell WL

4

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18 February 2018

Bullock R

5

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18 February 2018

publication date

1 April 1998

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18 February 2018

Journal of Clinical Neuroscience

published in

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18 February 2018

volume

5

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18 February 2018

issue

2

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18 February 2018

page(s)

193-202

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18 February 2018

Synaptic release of excitatory amino acid neurotransmitter mediates anoxic neuronal death

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Elevation of the Extracellular Concentrations of Glutamate and Aspartate in Rat Hippocampus During Transient Cerebral Ischemia Monitored by Intracerebral Microdialysis

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The role of excitatory amino acids and NMDA receptors in traumatic brain injury

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"Epileptic" brain damage is replicated qualitatively in the rat hippocampus by central injection of glutamate or aspartate but not by GABA or acetylcholine

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Ionic dependence of glutamate neurotoxicity

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Differential sensitivity of rat cerebellar cells in vitro to the neurotoxic effects of excitatory amino acid analogues

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Light- and electron-microscopic changes in central nervous tissue after electrophoretic injection of glutamate

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Hundred-fold increase in neuronal vulnerability to glutamate toxicity in astrocyte-poor cultures of rat cerebral cortex

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Glutamate uptake disguises neurotoxic potency of glutamate agonists in cerebral cortex in dissociated cell culture

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Neurotoxicity of L-glutamate and DL-threo-3-hydroxyaspartate in the rat striatum

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Delayed neuronal death in the rat hippocampus following transient forebrain ischemia

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Pathophysiology and treatment of focal cerebral ischemia. Part I: Pathophysiology

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Enhancement of NMDA receptor-mediated neurotoxicity in the hippocampal slice by depolarization and ischemia

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Identifiers

DOI

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18 February 2018

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18 February 2018