(Q49064534)

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Lithium sensitizes tumor cells in an NF-kappa B-independent way to caspase activation and apoptosis induced by tumor necrosis factor (TNF). Evidence for a role of the TNF receptor-associated death domain protein

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Lithium sensitizes tumor cells in an NF-kappa B-independent way to caspase activation and apoptosis induced by tumor necrosis factor (TNF). Evidence for a role of the TNF receptor-associated death domain protein (English)

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

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Schotte P

1

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Van Loo G

2

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Carpentier I

3

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Vandenabeele P

4

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

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7 May 2001

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Journal of Biological Chemistry

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

276

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

28

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

25939-25945

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Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

Molecular mechanisms of tumor necrosis factor-induced cytotoxicity. What we do understand and what we do not.

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Lithium chloride potentiates tumor necrosis factor-mediated cytotoxicity in vitro and in vivo

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More than one way to die: apoptosis, necrosis and reactive oxygen damage

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Caspases: enemies within

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FLICE, a novel FADD-homologous ICE/CED-3-like protease, is recruited to the CD95 (Fas/APO-1) death--inducing signaling complex

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Molecular determinants of the caspase-promoting activity of Smac/DIABLO and its role in the death receptor pathway

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Smac, a mitochondrial protein that promotes cytochrome c-dependent caspase activation by eliminating IAP inhibition

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p38 and extracellular signal-regulated kinase mitogen-activated protein kinase pathways are required for nuclear factor-kappaB p65 transactivation mediated by tumor necrosis factor.

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Tumour necrosis factor-induced necrosis versus anti-Fas-induced apoptosis in L929 cells.

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Tight control of gene expression in mammalian cells by tetracycline-responsive promoters

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Functional analysis of Fas signaling in vivo using synthetic inducers of dimerization

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A20 inhibits NF-kappaB activation independently of binding to 14-3-3 proteins

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Lithium chloride potentiates tumor necrosis factor-induced and interleukin 1-induced cytokine and cytokine receptor expression

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An improved colorimetric assay for interleukin 2.

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Death receptor-induced apoptotic and necrotic cell death: differential role of caspases and mitochondria.

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Cytochrome c and dATP-dependent formation of Apaf-1/caspase-9 complex initiates an apoptotic protease cascade

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IAPs block apoptotic events induced by caspase-8 and cytochrome c by direct inhibition of distinct caspases

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Apoptosis induction by caspase-8 is amplified through the mitochondrial release of cytochrome c.

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Controlling Signal Transduction with Synthetic Ligands

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Inhibition of TNF-induced apoptosis by NF-kappa B.

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Signalling by CD95 and TNF receptors: not only life and death.

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TRADD-TRAF2 and TRADD-FADD interactions define two distinct TNF receptor 1 signal transduction pathways

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TNF-dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex

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The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

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Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

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Lithium inhibits glycogen synthase kinase-3 activity and mimics wingless signalling in intact cells

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The mood-stabilizing agent valproate inhibits the activity of glycogen synthase kinase-3.

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Indirubins inhibit glycogen synthase kinase-3 beta and CDK5/p25, two protein kinases involved in abnormal tau phosphorylation in Alzheimer's disease. A property common to most cyclin-dependent kinase inhibitors?

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The axis-inducing activity, stability, and subcellular distribution of beta-catenin is regulated in Xenopus embryos by glycogen synthase kinase 3

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Targeted disruption of the mouse Caspase 8 gene ablates cell death induction by the TNF receptors, Fas/Apo1, and DR3 and is lethal prenatally.

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Identifiers

10.1074/JBC.M104014200

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

18 February 2018

http://europepmc.org/abstract/MED/11342564

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