(Q49138361)

18 February 2018

title

The expression of cocaine sensitization is not prevented by MK-801 or ibotenic acid lesions of the medial prefrontal cortex. (English)

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18 February 2018

author name string

Li Y

series ordinal

1

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18 February 2018

Wolf ME

series ordinal

2

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18 February 2018

White FJ

series ordinal

3

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18 February 2018

language of work or name

English

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publication date

1 October 1999

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18 February 2018

Behavioural Brain Research

published in

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18 February 2018

volume

104

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18 February 2018

issue

1-2

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18 February 2018

page(s)

119-125

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18 February 2018

Possible involvement of medial prefrontal cortex in amphetamine-induced sensitization of mesolimbic dopamine function

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Alterations in excitatory amino acid-mediated regulation of midbrain dopaminergic neurones induced by chronic psychostimulant administration and stress: relevance to behavioural sensitization and drug addiction.

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NMDA receptor inhibition prevents tolerance to cocaine

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Antagonist of N-methyl-D-aspartate receptors partially prevent the development of cocaine sensitization.

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Augmentation of the neurochemical effects of cocaine in the ventral striatum and medial prefrontal cortex following preexposure to amphetamine, but not nicotine: an in vivo microdialysis study.

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7 January 2021

Inhibition of cocaine sensitization by MK-801, a noncompetitive N-methyl-D-aspartate (NMDA) receptor antagonist: evaluation by ambulatory activity in mice.

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MK-801 prevents the development of behavioral sensitization during repeated morphine administration

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Dopamine transmission in the initiation and expression of drug- and stress-induced sensitization of motor activity

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Blockade of "reverse tolerance" to cocaine and amphetamine by MK-801.

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Amphetamine behavioral sensitization and the excitatory amino acids

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DNQX blockade of amphetamine behavioral sensitization

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Excitatory amino acids and the actions of cocaine

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Cocaine behavioral sensitization and the excitatory amino acids.

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A dopaminergic-glutamatergic basis for the action of amphetamine and cocaine.

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A persistent postsynaptic modification mediates long-term potentiation in the hippocampus

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Inhibition by MK-801 of cocaine-induced sensitization, conditioned place preference, and dopamine-receptor supersensitivity in mice.

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Metabotropic glutamate receptors are necessary for sensitization by amphetamine.

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Effects of the AMPA receptor antagonist NBQX on the development and expression of behavioral sensitization to cocaine and amphetamine.

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7 January 2021

Ibotenic acid lesions of prefrontal cortex do not prevent expression of behavioral sensitization to amphetamine.

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7 January 2021

Can the "state-dependency" hypothesis explain prevention of amphetamine sensitization in rats by NMDA receptor antagonists?

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R-(+)-HA-966, an antagonist for the glycine/NMDA receptor, prevents locomotor sensitization to repeated cocaine exposures.

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7 January 2021

Long-term potentiation differentially affects two components of synaptic responses in hippocampus

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7 January 2021

Repeated cocaine augments excitatory amino acid transmission in the nucleus accumbens only in rats having developed behavioral sensitization.

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7 January 2021

Ibotenic acid lesions of the dorsal prefrontal cortex disrupt the expression of behavioral sensitization to cocaine.

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7 January 2021

L-NAME and MK-801 attenuate sensitization to the locomotor-stimulating effect of cocaine.

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7 January 2021

The neural basis of drug craving: an incentive-sensitization theory of addiction

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7 January 2021

Sensitization to cocaine's motor activating properties produced by electrical kindling of the medial prefrontal cortex but not of the hippocampus

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Topographical organization of the efferent projections of the medial prefrontal cortex in the rat: an anterograde tract-tracing study with Phaseolus vulgaris leucoagglutinin

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Prefrontal cortical efferents in the rat synapse on unlabeled neuronal targets of catecholamine terminals in the nucleus accumbens septi and on dopamine neurons in the ventral tegmental area

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Cortical regulation of subcortical dopamine release: mediation via the ventral tegmental area

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7 January 2021

Chronic administration of (+)-amphetamine alters the reactivity of midbrain dopaminergic neurons to prefrontal cortex stimulation in the rat

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7 January 2021

The development of cocaine-induced behavioral sensitization is affected by discrete quinolinic acid lesions of the prelimbic medial prefrontal cortex.

1 reference

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7 January 2021

Repeated administration of MK-801 produces sensitization to its own locomotor stimulant effects but blocks sensitization to amphetamine.

1 reference

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7 January 2021

Coadministration of MK-801 with amphetamine, cocaine or morphine prevents rather than transiently masks the development of behavioral sensitization.

1 reference

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7 January 2021

Effects of lesions of prefrontal cortex, amygdala, or fornix on behavioral sensitization to amphetamine: comparison with N-methyl-D-aspartate antagonists

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7 January 2021

The role of excitatory amino acids in behavioral sensitization to psychomotor stimulants

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7 January 2021

10.1016/S0166-4328(99)00060-1

Identifiers

DOI

1 reference

18 February 2018

PubMed ID

11125730

1 reference

18 February 2018