(Q50508758)

14 March 2018

title

Prolonged exposure to hyperbaric oxygen induces neuronal damage in primary rat cortical cultures (English)

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14 March 2018

author name string

K L Huang

1

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14 March 2018

J N Wu

2

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14 March 2018

H C Lin

3

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14 March 2018

S P Mao

4

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14 March 2018

B Kang

5

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14 March 2018

F J Wan

6

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14 March 2018

language of work or name

English

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publication date

1 November 2000

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14 March 2018

published in

Neuroscience Letters

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14 March 2018

volume

293

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14 March 2018

issue

3

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14 March 2018

page(s)

159-162

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14 March 2018

Blockade of hyperbaric oxygen induced seizures by excitatory amino acid antagonists

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Generation of reactive oxygen species, release of L-glutamate and activation of caspases are required for oxygen-induced apoptosis of embryonic hippocampal neurons in culture.

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Depression of cortical Na+, K+-ATPase acticvity in rats exposed to hyperbaric oxygen

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Amino acids and ammonia in the cerebral cortex, the corpus striatum and the brain stem of the mouse prior to the onset and after a seizure induced by hyperbaric oxygen

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Excitatory amino acid release and free radical formation may cooperate in the genesis of ischemia-induced neuronal damage.

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Oxygen toxicity induces apoptosis in neuronal cells.

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Inactivation of mammalian brain glutamine synthetase by oxygen radicals

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Role of nitric oxide in neurodegenerative diseases.

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Correlation between decreases in brain gamma-aminobutyric acid levels and susceptibility to convulsions induced by hyperbaric oxygen

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In vivo evidence of hydroxyl radical formation induced by elevation of extracellular glutamate after cerebral ischemia in the cortex of anesthetized rats

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Cerebral amino acid, norepinephrine and nitric oxide metabolism in CNS oxygen toxicity

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Identifiers

DOI

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14 March 2018

1 reference

14 March 2018