(Q50517258)

14 March 2018

title

Evidence of a central role for p38 map kinase induction of tumor necrosis factor alpha in pancreatitis-associated pulmonary injury. (English)

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14 March 2018

author

Kevin J. Tracey

5

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14 March 2018

author name string

Yang J

1

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14 March 2018

Murphy C

2

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14 March 2018

Denham W

3

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14 March 2018

Botchkina G

4

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14 March 2018

Norman J

6

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14 March 2018

language of work or name

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1 August 1999

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14 March 2018

published in

Surgery

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14 March 2018

volume

126

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14 March 2018

issue

2

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14 March 2018

page(s)

216-222

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14 March 2018

Death due to acute pancreatitis. A retrospective analysis of 405 autopsy cases

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High bronchoalveolar levels of tumor necrosis factor and its inhibitors, interleukin-1, interferon, and elastase, in patients with adult respiratory distress syndrome after trauma, shock, or sepsis

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Acute pancreatitis induces intrapancreatic tumor necrosis factor gene expression

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Gene targeting demonstrates additive detrimental effects of interleukin 1 and tumor necrosis factor during pancreatitis

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Tissue-specific cytokine production during experimental acute pancreatitis. A probable mechanism for distant organ dysfunction

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Jun N-terminal kinase/stress-activated protein kinase (JNK/SAPK) is required for lipopolysaccharide stimulation of tumor necrosis factor alpha (TNF-alpha) translation: glucocorticoids inhibit TNF-alpha translation by blocking JNK/SAPK.

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Protein kinase C-alpha modulates lipopolysaccharide-induced functions in a murine macrophage cell line

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Suppression of proinflammatory cytokines in monocytes by a tetravalent guanylhydrazone

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CNI-1493 inhibits monocyte/macrophage tumor necrosis factor by suppression of translation efficiency.

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The critical role of p38 MAP kinase in T cell HIV-1 replication.

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The physiologic consequences of macrophage pacification during severe acute pancreatitis

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Macrophage pacification reduces rodent pancreatitis-induced hepatocellular injury through down-regulation of hepatic tumor necrosis factor ? and interleukin-1?

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Evidence for an unknown component of pancreatic ascites that induces adult respiratory distress syndrome through an interleukin-1 and tumor necrosis factor-dependent mechanism

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Targeted disruption of the beta-chemokine receptor CCR1 protects against pancreatitis-associated lung injury

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Evans blue dye as a marker of albumin clearance in cultured endothelial monolayer and isolated lung

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Respiratory mechanics and lung morphometry in severe pancreatitis-associated acute lung injury in rats

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Distribution and characterization of tumor necrosis factor-alpha-like immunoreactivity in the murine central nervous system

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Identifiers

DOI

1 reference

14 March 2018

1 reference

14 March 2018