(Q51070660)

28 March 2018

title

RIPK3-mediated necroptosis regulates cardiac allograft rejection. (English)

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28 March 2018

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A Pavlosky

1

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28 March 2018

A Lau

2

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Y Su

3

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28 March 2018

D Lian

4

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28 March 2018

X Huang

5

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Z Yin

6

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A Haig

7

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A M Jevnikar

8

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Z-X Zhang

9

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language of work or name

English

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publication date

1 July 2014

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28 March 2018

American Journal of Transplantation

published in

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28 March 2018

volume

14

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28 March 2018

issue

8

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28 March 2018

page(s)

1778-1790

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28 March 2018

The Registry of the International Society for Heart and Lung Transplantation: 29th adult lung and heart-lung transplant report-2012.

cites work

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Necroptosis: the release of damage-associated molecular patterns and its physiological relevance

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Tumour necrosis factor-induced necrosis versus anti-Fas-induced apoptosis in L929 cells.

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Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

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TNF receptors differentially signal and are differentially expressed and regulated in the human heart.

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Role of TNFalpha in early chemokine production and leukocyte infiltration into heart allografts

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Anti-tumor necrosis factor antibody enhances allograft survival in rats.

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Molecular mechanisms of necroptosis: an ordered cellular explosion

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RIP kinases at the crossroads of cell death and survival

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Receptor interacting protein kinase-3 determines cellular necrotic response to TNF-alpha

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Mixed lineage kinase domain-like protein mediates necrosis signaling downstream of RIP3 kinase

21 January 2018

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The pseudokinase MLKL mediates necroptosis via a molecular switch mechanism

21 January 2018

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Interferon-induced RIP1/RIP3-mediated necrosis requires PKR and is licensed by FADD and caspases

21 January 2018

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Release of chromatin protein HMGB1 by necrotic cells triggers inflammation

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1 reference

Pattern recognition receptors and inflammation

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1 reference

Critical role of the Toll-like receptor signal adaptor protein MyD88 in acute allograft rejection

21 January 2018

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Mechanisms underlying blockade of allograft acceptance by TLR ligands

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Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

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FADD prevents RIP3-mediated epithelial cell necrosis and chronic intestinal inflammation.

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Absence of receptor interacting protein kinase 3 prevents ethanol-induced liver injury

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1 reference

Necrostatin: a potentially novel cardioprotective agent?

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1 reference

Rip1 (receptor-interacting protein kinase 1) mediates necroptosis and contributes to renal ischemia/reperfusion injury

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1 reference

Inhibition of RIP1-dependent necrosis prevents adverse cardiac remodeling after myocardial ischemia-reperfusion in vivo.

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1 reference

RIPK3-mediated necroptosis promotes donor kidney inflammatory injury and reduces allograft survival.

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Kinase RIP3 is dispensable for normal NF-kappa Bs, signaling by the B-cell and T-cell receptors, tumor necrosis factor receptor 1, and Toll-like receptors 2 and 4.

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NK cells induce apoptosis in tubular epithelial cells and contribute to renal ischemia-reperfusion injury

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Molecular and cellular properties of PECAM-1 (endoCAM/CD31): a novel vascular cell-cell adhesion molecule

21 January 2018

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Cloning and expression of a cDNA encoding mouse endoglin, an endothelial cell TGF-beta ligand

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A death-domain-containing receptor that mediates apoptosis

21 January 2018

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Cloning and expression of cDNAs for two distinct murine tumor necrosis factor receptors demonstrate one receptor is species specific

21 January 2018

1 reference

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21 January 2018

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The death domain kinase RIP mediates the TNF-induced NF-kappaB signal

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21 January 2018

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21 January 2018

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21 January 2018

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Identification of RIP1 kinase as a specific cellular target of necrostatins

21 January 2018

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Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

21 January 2018

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Necroptosis, necrosis and secondary necrosis converge on similar cellular disintegration features.

21 January 2018

1 reference

Extracellular hmgb1 functions as an innate immune-mediator implicated in murine cardiac allograft acute rejection

21 January 2018

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Toll-like receptors activate programmed necrosis in macrophages through a receptor-interacting kinase-3-mediated pathway

21 January 2018

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Toll-like receptor 3-mediated necrosis via TRIF, RIP3, and MLKL.

21 January 2018

1 reference

The RIP1-kinase inhibitor necrostatin-1 prevents osmotic nephrosis and contrast-induced AKI in mice

21 January 2018

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The nuclear protein HMGB1 is secreted by monocytes via a non-classical, vesicle-mediated secretory pathway

21 January 2018

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21 January 2018

Identifiers

DOI

10.1111/AJT.12779

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28 March 2018

PubMed ID

24984764

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28 March 2018