(Q51384261)

4 April 2018

title

Effects of cocaine self-administration on plasma corticosterone in rats: relationship to hippocampal type II glucocorticoid receptors. (English)

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4 April 2018

author name string

Mantsch JR

series ordinal

1

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4 April 2018

Goeders NE

series ordinal

2

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4 April 2018

publication date

1 May 2000

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4 April 2018

Progress in Neuro-Psychopharmacology & Biological Psychiatry

published in

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4 April 2018

volume

24

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4 April 2018

issue

4

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4 April 2018

page(s)

633-646

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4 April 2018

Effects of intravenous cocaine on plasma cortisol and prolactin in human cocaine abusers.

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The role of corticotropin-releasing factor and corticosterone in stress- and cocaine-induced relapse to cocaine seeking in rats.

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Characterization of a monoclonal antibody to the rat liver glucocorticoid receptor

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Role of corticosterone in intravenous cocaine self-administration in rats.

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Effects of surgical and pharmacological adrenalectomy on the initiation and maintenance of intravenous cocaine self-administration in rats.

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A neuroendocrine role in cocaine reinforcement

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Ketoconazole reduces low dose cocaine self-administration in rats

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Differences in extracellular dopamine concentrations in the nucleus accumbens during response-dependent and response-independent cocaine administration in the rat.

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Drug dependence: stress and dysregulation of brain reward pathways

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Cleavage of Structural Proteins during the Assembly of the Head of Bacteriophage T4

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Hippocampal type I and type II corticosteroid receptor affinities are reduced in rats predisposed to develop amphetamine self-administration

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Ketoconazole blocks the stress-induced reinstatement of cocaine-seeking behavior in rats: relationship to the discriminative stimulus effects of cocaine.

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Cocaine effects on pulsatile secretion of anterior pituitary, gonadal, and adrenal hormones

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Cocaine induced secretion of ACTH, beta-endorphin, and corticosterone

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Pathophysiological basis of vulnerability to drug abuse: role of an interaction between stress, glucocorticoids, and dopaminergic neurons

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Cocaine stimulates adrenocorticotropin (ACTH) secretion through a corticotropin-releasing factor (CRF)-mediated mechanism

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The cocaine-induced elevation of plasma corticosterone is mediated by endogenous corticotropin-releasing factor (CRF) in rats.

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Neuroendocrine-related effects of long-term, 'binge' cocaine administration: diminished individual differences in stress-induced corticosterone response

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Hippocampal glucocorticoid receptor mRNA is up-regulated by acute and down-regulated by chronic amphetamine treatment

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Glucocorticoid feedback regulation of adrenocortical responses to neural stimuli: role of CRF-41 and corticosteroid type I and type II receptors

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Identifiers

DOI

1 reference

4 April 2018

1 reference

4 April 2018