(Q51584941)

7 April 2018

title

Negative regulation of the RelA/p65 transactivation function by the product of the DEK proto-oncogene. (English)

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7 April 2018

author name string

Morgan Sammons

1

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7 April 2018

Shan Shan Wan

2

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7 April 2018

Nancy L Vogel

3

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7 April 2018

Edwin J Mientjes

4

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7 April 2018

Gerard Grosveld

5

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7 April 2018

Brian P Ashburner

6

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7 April 2018

language of work or name

English

0 references

publication date

7 July 2006

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7 April 2018

Journal of Biological Chemistry

published in

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7 April 2018

volume

281

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7 April 2018

issue

37

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7 April 2018

page(s)

26802-26812

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7 April 2018

Regulation of inducible gene expression by the transcription factor NF-kappaB.

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The p65 (RelA) subunit of NF-kappaB interacts with the histone deacetylase (HDAC) corepressors HDAC1 and HDAC2 to negatively regulate gene expression

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DEK, an autoantigen involved in a chromosomal translocation in acute myelogenous leukemia, binds to the HIV-2 enhancer

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Characterization of the Stat5 protease

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Ineffectiveness of histone deacetylase inhibitors to induce apoptosis involves the transcriptional activation of NF-kappa B through the Akt pathway

21 January 2018

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RING Finger Protein AO7 Supports NF-κB-mediated Transcription by Interacting with the Transactivation Domain of the p65 Subunit

21 January 2018

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IkappaB kinase alpha-mediated derepression of SMRT potentiates acetylation of RelA/p65 by p300.

21 January 2018

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Conditional knockout mice reveal distinct functions for the global transcriptional coactivators CBP and p300 in T-cell development

21 January 2018

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21 January 2018

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Interaction of histone acetylases and deacetylases in vivo

21 January 2018

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Regulation of histone acetyltransferase activity of p300 and PCAF by proto-oncogene protein DEK.

21 January 2018

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21 January 2018

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Structure-specific binding of the proto-oncogene protein DEK to DNA.

21 January 2018

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21 January 2018

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21 January 2018

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Coordination of a transcriptional switch by HMGI(Y) acetylation

21 January 2018

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21 January 2018

Identifiers

DOI

10.1074/JBC.M600915200

1 reference

7 April 2018

1 reference

7 April 2018