(Q51612396)

English

Daidzein supplementation prevents non-alcoholic fatty liver disease through alternation of hepatic gene expression profiles and adipocyte metabolism.

scientific article published on 14 December 2010

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scholarly article

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

Daidzein supplementation prevents non-alcoholic fatty liver disease through alternation of hepatic gene expression profiles and adipocyte metabolism. (English)

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

1 reference

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inferred from title

metabolic dysfunction–associated steatotic liver disease

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based on heuristic

inferred from title

non-alcoholic fatty liver

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based on heuristic

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author name string

M-H Kim

1

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

J-S Park

2

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

J-W Jung

3

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

K-W Byun

4

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

K-S Kang

5

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

Y-S Lee

6

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

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publication date

14 December 2010

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

International Journal of Obesity

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

35

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

8

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Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

1019-1030

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

The natural history of nonalcoholic fatty liver disease: a population-based cohort study

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Glucose and cAMP regulate the L-type pyruvate kinase gene by phosphorylation/dephosphorylation of the carbohydrate response element binding protein

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Role of ChREBP in hepatic steatosis and insulin resistance.

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The liver X receptor (LXR) and hepatic lipogenesis. The carbohydrate-response element-binding protein is a target gene of LXR

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Mechanism for fatty acid "sparing" effect on glucose-induced transcription: regulation of carbohydrate-responsive element-binding protein by AMP-activated protein kinase

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cAMP opposes the glucose-mediated induction of the L-PK gene by preventing the recruitment of a complex containing ChREBP, HNF4alpha, and CBP.

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c-Jun N-terminal kinase (JNK) mediates feedback inhibition of the insulin signaling cascade

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Serine phosphorylation of insulin receptor substrate 1 by inhibitor kappa B kinase complex.

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Altered constitutive expression of fatty acid-metabolizing enzymes in mice lacking the peroxisome proliferator-activated receptor alpha (PPARalpha)

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Long-chain fatty acids regulate liver carnitine palmitoyltransferase I gene (L-CPT I) expression through a peroxisome-proliferator-activated receptor alpha (PPARalpha)-independent pathway

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The fat-derived hormone adiponectin alleviates alcoholic and nonalcoholic fatty liver diseases in mice

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Enhanced carbon tetrachloride-induced liver fibrosis in mice lacking adiponectin

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Identifiers

10.1038/IJO.2010.256

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

8 April 2018

http://europepmc.org/abstract/MED/21157426

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