(Q51692419)

9 April 2018

title

Toll-like receptor-4 is involved in eliciting an LPS-induced oxidative burst in neutrophils. (English)

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9 April 2018

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Katharina A Remer

1

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9 April 2018

Marija Brcic

series ordinal

2

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9 April 2018

Thomas W Jungi

series ordinal

3

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9 April 2018

publication date

1 January 2003

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9 April 2018

published in

immunology Letters

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9 April 2018

volume

85

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9 April 2018

issue

1

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9 April 2018

page(s)

75-80

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9 April 2018

The immune system evolved to discriminate infectious nonself from noninfectious self

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RICK/Rip2/CARDIAK mediates signalling for receptors of the innate and adaptive immune systems

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LPS directly induces oxygen radical production in human monocytes via LPS binding protein and CD14.

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Lucigenin-dependent chemiluminescence as a new assay for NAD(P)H-oxidase activity in particulate fractions of human polymorphonuclear leukocytes

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Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

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Involvement of protein kinase C and tyrosin kinase in tumoricidal activation of macrophage induced by Streptococcus pneumoniae type II capsular polysaccharide

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Induction of oxidative burst response in human neutrophils by immune complexes made in vitro of lipopolysaccharide and hyperimmune serum from chronically infected patients

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Lipopolysaccharide-binding protein- and CD14-dependent activation of mitogen-activated protein kinase p38 by lipopolysaccharide in human neutrophils is associated with priming of respiratory burst.

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Increased production of superoxide anion by macrophages exposed in vitro to muramyl dipeptide or lipopolysaccharide

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Identifiers

DOI

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9 April 2018

1 reference

9 April 2018