(Q51770946)

12 April 2018

title

BM stromal cells ameliorate experimental autoimmune myasthenia gravis by altering the balance of Th cells through the secretion of IDO. (English)

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12 April 2018

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Qing-fei Kong

1

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12 April 2018

Bo Sun

2

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12 April 2018

Guang-you Wang

3

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12 April 2018

Dong-xu Zhai

4

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12 April 2018

Li-li Mu

5

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12 April 2018

Dan-dan Wang

6

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12 April 2018

Jing-hua Wang

7

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12 April 2018

Rui Li

8

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12 April 2018

Hu-lun Li

9

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12 April 2018

language of work or name

English

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publication date

1 March 2009

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12 April 2018

European Journal of Immunology

published in

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12 April 2018

volume

39

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12 April 2018

issue

3

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12 April 2018

page(s)

800-809

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12 April 2018

Multilineage potential of adult human mesenchymal stem cells

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Bone marrow stromal cells generate muscle cells and repair muscle degeneration.

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Role for interferon-gamma in the immunomodulatory activity of human bone marrow mesenchymal stem cells.

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Human mesenchymal stem cells modulate B-cell functions

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Role of IL-17 and regulatory T lymphocytes in a systemic autoimmune disease

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Mesenchymal stem cells ameliorate experimental autoimmune encephalomyelitis inducing T-cell anergy.

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Human mesenchymal stem cells alter antigen-presenting cell maturation and induce T-cell unresponsiveness

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Human bone marrow stromal cells inhibit allogeneic T-cell responses by indoleamine 2,3-dioxygenase-mediated tryptophan degradation

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Immunoregulation in experimental autoimmune myasthenia gravis--about T cells, antibodies, and endplates

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Experimental autoimmune myasthenia: A model of myasthenia gravis in rats and guinea pigs

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Interferon-gamma-modified dendritic cells suppress B cell function and ameliorate the development of experimental autoimmune myasthenia gravis

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Indoleamine 2,3-dioxygenase in lung dendritic cells promotes Th2 responses and allergic inflammation

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Functional yet balanced reactivity to Candida albicans requires TRIF, MyD88, and IDO-dependent inhibition of Rorc.

21 January 2018

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Bone marrow mesenchymal stem cells inhibit the response of naive and memory antigen-specific T cells to their cognate peptide.

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T Cells and Cytokines in the Pathogenesis of Acquired Myasthenia Gravis

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Myasthenia gravis-like syndrome induced by expression of interferon gamma in the neuromuscular junction.

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Interferon γ (IFN-γ) Is Necessary for the Genesis of Acetylcholine Receptor–induced Clinical Experimental Autoimmune Myasthenia gravis in Mice

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Breakdown of tolerance to a self-peptide of acetylcholine receptor alpha-subunit induces experimental myasthenia gravis in rats

21 January 2018

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In vitro preprogramming of marrow stromal cells for myocardial regeneration

21 January 2018

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Bone marrow mesenchymal stem cells induce division arrest anergy of activated T cells

21 January 2018

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Human mesenchymal stem cells inhibit differentiation and function of monocyte-derived dendritic cells

21 January 2018

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7 January 2021

Mesenchymal stem cells inhibit and stimulate mixed lymphocyte cultures and mitogenic responses independently of the major histocompatibility complex

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7 January 2021

CD4+ T and B cells cooperate in the immunoregulation of Experimental Autoimmune Myasthenia Gravis

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7 January 2021

IL-12 is involved in the induction of experimental autoimmune myasthenia gravis, an antibody-mediated disease

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7 January 2021

Cooperation of Invariant NKT Cells and CD4+CD25+ T Regulatory Cells in the Prevention of Autoimmune Myasthenia

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7 January 2021

IL-17 plays an important role in the development of experimental autoimmune encephalomyelitis

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7 January 2021

IL-6-gp130-STAT3 in T cells directs the development of IL-17+ Th with a minimum effect on that of Treg in the steady state

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https://api.crossref.org/works/10.1002%2FEJI.200838729

7 January 2021

Identifiers

DOI

10.1002/EJI.200838729

1 reference

12 April 2018

1 reference

12 April 2018