(Q52101085)

22 April 2018

title

The chemotactic and mitogenic responses of vascular smooth muscle cells to insulin-like growth factor-I require the activation of ERK1/2. (English)

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22 April 2018

1 reference

Cunming Duan

1

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22 April 2018

publication date

1 August 2003

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22 April 2018

Molecular and Cellular Endocrinology

published in

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22 April 2018

volume

206

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22 April 2018

issue

1-2

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22 April 2018

page(s)

75-83

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22 April 2018

Regulation of vascular smooth muscle cell apoptosis. Modulation of bad by a phosphatidylinositol 3-kinase-dependent pathway

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The insulin-like growth factor axis: A review of atherosclerosis and restenosis

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Platelet-derived growth factor inhibits smooth muscle cell adhesion to fibronectin by ERK-dependent and ERK-independent pathways.

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Insulin-like growth factor-I and platelet-derived growth factor-BB induce directed migration of human arterial smooth muscle cells via signaling pathways that are distinct from those of proliferation

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Platelet-derived growth factor. Distinct signal transduction pathways associated with migration versus proliferation

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The pathogenesis of atherosclerosis: a perspective for the 1990s

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Regulation of vascular smooth muscle cell insulin-like growth factor I receptors by phosphorothioate oligonucleotides. Effects on cell growth and evidence that sense targeting at the ATG site increases receptor expression

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Inhibition of vascular smooth muscle cell growth through antisense transcription of a rat insulin-like growth factor I receptor cDNA.

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Specifying the cellular responses to IGF signals: roles of IGF-binding proteins

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Insulin-like growth factor-I (IGF-I) regulates IGF-binding protein-5 synthesis through transcriptional activation of the gene in aortic smooth muscle cells

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Insulin-like growth factor (IGF)-I regulates IGF-binding protein-5 gene expression through the phosphatidylinositol 3-kinase, protein kinase B/Akt, and p70 S6 kinase signaling pathway

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Phosphatidylinositol 3-kinase is required for insulin-like growth factor-I-induced vascular smooth muscle cell proliferation and migration

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Identification of a novel inhibitor of mitogen-activated protein kinase kinase

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Angiotensin II inhibits insulin signaling in aortic smooth muscle cells at multiple levels. A potential role for serine phosphorylation in insulin/angiotensin II crosstalk

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Mitogen-activated protein kinase activation is involved in platelet-derived growth factor-directed migration by vascular smooth muscle cells

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7 January 2021

Platelet-derived growth factor stimulates protein kinase A through a mitogen-activated protein kinase-dependent pathway in human arterial smooth muscle cells

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Insulin-like growth factor-I-mediated neurite outgrowth in vitro requires mitogen-activated protein kinase activation.

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The fumagillin analogue TNP-470 inhibits DNA synthesis of vascular smooth muscle cells stimulated by platelet-derived growth factor and insulin-like growth factor-I. Possible involvement of cyclin-dependent kinase 2

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Molecular and cellular aspects of the insulin-like growth factor I receptor

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Insulin-like Growth Factor 1 Inhibits Apoptosis Using the Phosphatidylinositol 3′-Kinase and Mitogen-activated Protein Kinase Pathways

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Insulin-specific activation of S6 kinase and its desensitization in cultured rat vascular smooth muscle cells

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7 January 2021

The synergistic effect of PDGF-AA and IGF-1 on VSMC proliferation might be explained by the differential activation of their intracellular signaling pathways

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The IRS-signalling system: a network of docking proteins that mediate insulin action

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Insulin-like growth factor II plays a central role in atherosclerosis in a mouse model.

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7 January 2021

10.1016/S0303-7207(03)00212-0

Identifiers

DOI

1 reference

22 April 2018

1 reference

22 April 2018