(Q52170156)

26 April 2018

title

Ceramide selectively inhibits apoptosis-associated events in NGF-deprived sympathetic neurons. (English)

1 reference

26 April 2018

1 reference

Nair P

1

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26 April 2018

Tammariello SP

series ordinal

2

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26 April 2018

Estus S

series ordinal

3

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26 April 2018

publication date

1 February 2000

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26 April 2018

Cell Death & Differentiation

published in

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26 April 2018

volume

7

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26 April 2018

issue

2

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26 April 2018

page(s)

207-214

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26 April 2018

https://scigraph.springernature.com/pub.10.1038/sj.cdd.4400628

exact match

0 references

cites work

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Phosphorylation of Raf by ceramide-activated protein kinase

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Requirement for ceramide-initiated SAPK/JNK signalling in stress-induced apoptosis.

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Rapid suppression of free radical formation by nerve growth factor involves the mitogen-activated protein kinase pathway

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Temporal analysis of events associated with programmed cell death (apoptosis) of sympathetic neurons deprived of nerve growth factor

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Characterization of apoptosis in cultured rat sympathetic neurons after nerve growth factor withdrawal

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Altered gene expression in neurons during programmed cell death: identification of c-jun as necessary for neuronal apoptosis

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Genetic and metabolic status of NGF-deprived sympathetic neurons saved by an inhibitor of ICE family proteases

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Inhibitors of protein synthesis and RNA synthesis prevent neuronal death caused by nerve growth factor deprivation

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Ordering the cell death pathway. Differential effects of BCL2, an interleukin-1-converting enzyme family protease inhibitor, and other survival agents on JNK activation in serum/nerve growth factor-deprived PC12 cells

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The small GTP-binding protein Cdc42 is required for nerve growth factor withdrawal-induced neuronal death

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Regulation of the phagocyte respiratory burst by small GTP-binding proteins

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Ceramide prevents neuronal programmed cell death induced by nerve growth factor deprivation

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Ceramide activates the stress-activated protein kinases

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Ceramide can induce cell death in sensory neurons, whereas ceramide analogues and sphingosine promote survival

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Mitogen-activated protein kinase-independent pathways mediate the effects of nerve growth factor and cAMP on neuronal survival

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Superoxide dismutase delays neuronal apoptosis: a role for reactive oxygen species in programmed neuronal death

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A c-Jun dominant negative mutant protects sympathetic neurons against programmed cell death

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The antioxidant Trolox enhances the oxidation of 2',7'-dichlorofluorescin to 2',7'-dichlorofluorescein

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NADPH oxidase contributes directly to oxidative stress and apoptosis in nerve growth factor-deprived sympathetic neurons

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7 January 2021

Fas- or ceramide-induced apoptosis is mediated by a Rac1-regulated activation of Jun N-terminal kinase/p38 kinases and GADD153

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7 January 2021

Comparison between the timing of JNK activation, c-Jun phosphorylation, and onset of death commitment in sympathetic neurones.

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7 January 2021

Role of the Jun kinase pathway in the regulation of c-Jun expression and apoptosis in sympathetic neurons.

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Analysis of cell cycle-related gene expression in postmitotic neurons: selective induction of Cyclin D1 during programmed cell death

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7 January 2021

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7 January 2021

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7 January 2021

DNA damage and apoptosis in Alzheimer's disease: colocalization with c-Jun immunoreactivity, relationship to brain area, and effect of postmortem delay.

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7 January 2021

Global ischemia can cause DNA fragmentation indicative of apoptosis in rat brain

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7 January 2021

Aggregated amyloid-beta protein induces cortical neuronal apoptosis and concomitant "apoptotic" pattern of gene induction.

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7 January 2021

Identifiers

DOI

10.1038/SJ.CDD.4400628

1 reference

26 April 2018

Dimensions Publication ID

1022873515

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1 reference

26 April 2018