(Q52173561)

26 April 2018

title

The ontogeny of the neuroendocrine response to endotoxin. (English)

1 reference

26 April 2018

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Dent GW

1

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26 April 2018

Smith MA

series ordinal

2

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26 April 2018

Levine S

series ordinal

3

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26 April 2018

publication date

1 October 1999

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26 April 2018

number of pages

9

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Developmental Brain Research

inferred from page(s)

published in

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26 April 2018

volume

117

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26 April 2018

issue

1

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26 April 2018

page(s)

21-29

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26 April 2018

Corticotropin-releasing factor-producing neurons in the rat activated by interleukin-1.

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Lipopolysaccharide is able to bypass corticotrophin-releasing factor in affecting plasma ACTH and corticosterone levels: evidence from rats with lesions of the paraventricular nucleus

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Distribution of Fos-like immunoreactivity in the rat brain following intravenous lipopolysaccharide administration

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Endotoxin-induced corticotropin-releasing hormone gene expression in the hypothalamic paraventricular nucleus is mediated centrally by interleukin-1.

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In the mouse, the activation of the hypothalamic-pituitary-adrenal axis by a lipopolysaccharide (endotoxin) is mediated through interleukin-1.

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Interleukin-1 stimulates the secretion of hypothalamic corticotropin-releasing factor

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Hypothalamic-pituitary-adrenal activation following endotoxin administration in the developing rat: a CRH-mediated effect

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Maternal deprivation and stress induce immediate early genes in the infant rat brain

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Interleukin-1 stimulates corticotropin-releasing factor gene expression in rat hypothalamus

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Effects of maternal deprivation on the ACTH stress response in the infant rat

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Early vs. late maternal deprivation differentially alters the endocrine and hypothalamic responses to stress

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The pituitary-adrenocortical system of neonatal rats is responsive to stress throughout development in a time-dependent and stressor-specific fashion.

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Identifiers

DOI

1 reference

26 April 2018

1 reference

26 April 2018