(Q52174168)

26 April 2018

title

Interdigital cell death can occur through a necrotic and caspase-independent pathway. (English)

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26 April 2018

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3

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26 April 2018

author name string

Chautan M

1

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26 April 2018

Chazal G

2

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26 April 2018

Gruss P

4

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26 April 2018

Golstein P

5

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26 April 2018

language of work or name

English

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publication date

1 September 1999

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26 April 2018

published in

Current Biology

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26 April 2018

volume

9

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26 April 2018

page(s)

967-970

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26 April 2018

issue

17

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26 April 2018

Cell deaths in normal vertebrate ontogeny

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Apoptotic cell death in the mouse limb and its suppression in the hammertoe mutant

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Peptide inhibitors of the ICE protease family arrest programmed cell death of motoneurons in vivo and in vitro

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7 January 2021

Role of Ced-3/ICE-family proteases in staurosporine-induced programmed cell death

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Requirement for BMP signaling in interdigital apoptosis and scale formation.

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The immunophilin FKBP12 functions as a common inhibitor of the TGF beta family type I receptors.

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Inhibition of human caspases by peptide-based and macromolecular inhibitors

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Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3

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7 January 2021

Apaf1 (CED-4 homolog) regulates programmed cell death in mammalian development

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Apaf1 is required for mitochondrial pathways of apoptosis and brain development

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7 January 2021

Inhibition of Ced-3/ICE-related proteases does not prevent cell death induced by oncogenes, DNA damage, or the Bcl-2 homologue Bak

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7 January 2021

The apoptosis-necrosis paradox. Apoptogenic proteases activated after mitochondrial permeability transition determine the mode of cell death.

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Essential contribution of caspase 3/CPP32 to apoptosis and its associated nuclear changes

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Ligation of major histocompatability complex (MHC) class I molecules on human T cells induces cell death through PI-3 kinase-induced c-Jun NH2-terminal kinase activity: a novel apoptotic pathway distinct from Fas-induced apoptosis

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Do all programmed cell deaths occur via apoptosis?

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Molecular characterization of mitochondrial apoptosis-inducing factor

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7 January 2021

Inhibition of caspases increases the sensitivity of L929 cells to necrosis mediated by tumor necrosis factor.

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Caspase-independent cell killing by Fas-associated protein with death domain

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10.1016/S0960-9822(99)80425-4

Identifiers

DOI

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26 April 2018

PubMed ID

10508592

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26 April 2018