(Q52507854)

2 May 2018

title

Pharmacological correction of neonatal lethal hepatic dysfunction in a murine model of hereditary tyrosinaemia type I. (English)

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2 May 2018

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1

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2 May 2018

author name string

Lindstedt S

2

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2 May 2018

al-Dhalimy M

3

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2 May 2018

Kennaway NG

4

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2 May 2018

Papaconstantinou J

5

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2 May 2018

Torres-Ramos CA

6

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2 May 2018

Ou CN

7

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2 May 2018

Finegold M

8

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2 May 2018

language of work or name

English

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publication date

1 August 1995

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2 May 2018

published in

Nature Genetics

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2 May 2018

volume

10

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2 May 2018

issue

4

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2 May 2018

page(s)

453-460

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2 May 2018

https://scigraph.springernature.com/pub.10.1038/ng0895-453

exact match

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cites work

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Treatment of hereditary tyrosinaemia type I by inhibition of 4-hydroxyphenylpyruvate dioxygenase

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raf regulates the postnatal repression of the mouse alpha-fetoprotein gene at the posttranscriptional level

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Nucleotide sequence of a cDNA encoding murine fumarylacetoacetate hydrolase.

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Molecular characterization of the murine argininosuccinate synthetase locus.

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Neurologic crises in hereditary tyrosinemia

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Tyrosinaemia type I and hypertrophic obstructive cardiomyopathy

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Ionic calcium levels during pregnancy, at delivery and in the first hours of life

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Loss of fumarylacetoacetate hydrolase is responsible for the neonatal hepatic dysfunction phenotype of lethal albino mice.

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Complementation studies of lethal alleles in the mouse causing deficiencies of glucose-6-phosphatase, tyrosine aminotransferase, and serine dehydratase

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Evidence for regulatory genes on mouse chromosome 7 that affect the quantitative expression of proteins in the fetal and newborn liver

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Two genetically defined trans-acting loci coordinately regulate overlapping sets of liver-specific genes

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Rescue of mice homozygous for lethal albino deletions: implications for an animal model for the human liver disease tyrosinemia type 1

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Murine fumarylacetoacetate hydrolase (Fah) gene is disrupted by a neonatally lethal albino deletion that defines the hepatocyte-specific developmental regulation 1 (hsdr-1) locus

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Deficiency of an enzyme of tyrosine metabolism underlies altered gene expression in newborn liver of lethal albino mice.

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Trans regulation of the phosphoenolpyruvate carboxykinase (GTP) gene, identified by deletions in chromosome 7 of the mouse

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A lethal deletion on mouse chromosome 7 affects regulation of liver-cell-specific functions: posttranscriptional control of serum protein and transcriptional control of aldolase B synthesis

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Selective loss of a DNase I hypersensitive site upstream of the tyrosine aminotransferase gene in mice homozygous for lethal albino deletions

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Modulation of keratin 14 and alpha-fetoprotein expression during hepatic oval cell proliferation and liver regeneration

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Serum levels of oncofetal markers CA 125, CA 19-9, and alpha-fetoprotein in children with hereditary tyrosinemia type I

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Pediatric liver transplantation: the Montreal experience

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Liver transplantation in infants and children

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Diethylnitrosamine-induced metastasizing hepatocellular carcinomas in New Zealand white rabbits. A tumor model for clinical investigations

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Experimental WHV infection of woodchucks: an animal model of hepadnavirus-induced liver cancer

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Identifiers

DOI

10.1038/NG0895-453

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2 May 2018

Dimensions Publication ID

1044553748

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1 reference

2 May 2018