(Q52518316)

2 May 2018

title

Mode of action of the qcr9 and cat3 mutations in restoring the ability of Saccharomyces cerevisiae tps1 mutants to grow on glucose. (English)

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2 May 2018

main subject

Saccharomyces cerevisiae

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1

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2 May 2018

author name string

C Gancedo

series ordinal

2

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2 May 2018

publication date

1 December 1995

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2 May 2018

Molecular Genetics and Genomics

published in

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2 May 2018

volume

249

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2 May 2018

issue

6

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2 May 2018

page(s)

655-664

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2 May 2018

https://scigraph.springernature.com/pub.10.1007/bf00418035

exact match

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cites work

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A yeast homologue of the bovine lens fibre MIP gene family complements the growth defect of a Saccharomyces cerevisiae mutant on fermentable sugars but not its defect in glucose-induced RAS-mediated cAMP signalling

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GPD1, which encodes glycerol-3-phosphate dehydrogenase, is essential for growth under osmotic stress in Saccharomyces cerevisiae, and its expression is regulated by the high-osmolarity glycerol response pathway

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High-affinity glucose transport in Saccharomyces cerevisiae is under general glucose repression control

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The mutation DGT1-1 decreases glucose transport and alleviates carbon catabolite repression in Saccharomyces cerevisiae

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Trehalose-6-P synthase is dispensable for growth on glucose but not for spore germination in Schizosaccharomyces pombe

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The FPS1 gene product functions as a glycerol facilitator in the yeast Saccharomyces cerevisiae

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The CDC20 gene product of Saccharomyces cerevisiae, a beta-transducin homolog, is required for a subset of microtubule-dependent cellular processes

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Yeast sugar transporters

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THE ROLE OF AEROBIC PHOSPHORYLATION IN THE PASTEUR EFFECT

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12 December 2020

Identifiers

DOI

10.1007/BF00418035

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2 May 2018

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2 May 2018