(Q52962351)

9 May 2018

title

Th2 cells support intrinsic anti-inflammatory properties of the brain. (English)

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9 May 2018

author

Ulrike Gimsa

1

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9 May 2018

Susanne Wolf

2

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9 May 2018

author name string

Haas D

3

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9 May 2018

Bechmann I

4

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9 May 2018

Nitsch R

5

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9 May 2018

publication date

1 September 2001

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9 May 2018

Journal of Neuroimmunology

published in

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9 May 2018

volume

119

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9 May 2018

issue

1

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9 May 2018

page(s)

73-80

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9 May 2018

Copolymer 1 induces T cells of the T helper type 2 that crossreact with myelin basic protein and suppress experimental autoimmune encephalomyelitis

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A T helper cell 2 (Th2) immune response against non-self antigens modifies the cytokine profile of autoimmune T cells and protects against experimental allergic encephalomyelitis

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Requirement for CD40 ligand in costimulation induction, T cell activation, and experimental allergic encephalomyelitis.

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Fluorescent dye prelabelled microglial cells migrate into organotypic hippocampal slice cultures and ramify.

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Astrocytic factors deactivate antigen presenting cells that invade the central nervous system.

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Myelin basic protein-specific T helper 2 (Th2) cells cause experimental autoimmune encephalomyelitis in immunodeficient hosts rather than protect them from the disease

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Low avidity recognition of self-antigen by T cells permits escape from central tolerance.

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Local delivery of interleukin 4 by retrovirus-transduced T lymphocytes ameliorates experimental autoimmune encephalomyelitis.

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Ligation of CD40 on fibroblasts induces CD54 (ICAM-1) and CD106 (VCAM-1) up-regulation and IL-6 production and proliferation

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7 January 2021

10.1016/S0165-5728(01)00343-5

Identifiers

DOI

1 reference

9 May 2018

1 reference

9 May 2018