(Q53079362)

11 May 2018

title

Mice are unable to endogenously regenerate podocytes during the repair of immunotoxin-induced glomerular injury. (English)

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11 May 2018

author name string

Yoichi Miyazaki

1

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11 May 2018

Akihiro Shimizu

2

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11 May 2018

Iekuni Ichikawa

3

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11 May 2018

Tatsuo Hosoya

4

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11 May 2018

Ira Pastan

5

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11 May 2018

Taiji Matsusaka

6

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11 May 2018

language of work or name

English

0 references

publication date

8 December 2013

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11 May 2018

Nephrology Dialysis Transplantation

published in

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11 May 2018

volume

29

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11 May 2018

issue

5

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11 May 2018

page(s)

1005-1012

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11 May 2018

Cell biology of the glomerular podocyte

cites work

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Podocyte biology and response to injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Apical cell membranes are shed into urine from injured podocytes: a novel phenomenon of podocyte injury

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Bone marrow-derived cells contribute to podocyte regeneration and amelioration of renal disease in a mouse model of Alport syndrome.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Bone-marrow-derived stem cells repair basement membrane collagen defects and reverse genetic kidney disease

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Immigrating progenitor cells contribute to human podocyte turnover.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Recruitment of podocytes from glomerular parietal epithelial cells

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Regeneration of glomerular podocytes by human renal progenitors

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Renal progenitor cells contribute to hyperplastic lesions of podocytopathies and crescentic glomerulonephritis

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Parietal epithelial cells participate in the formation of sclerotic lesions in focal segmental glomerulosclerosis.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Genetic engineering of glomerular sclerosis in the mouse via control of onset and severity of podocyte-specific injury.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Podocyte injury damages other podocytes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Progressive glomerulosclerosis and enhanced renal accumulation of basement membrane components in mice transgenic for human immunodeficiency virus type 1 genes

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Early glomerular filtration defect and severe renal disease in podocin-deficient mice

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Ectopic expression of Thy-1 in the kidneys of transgenic mice induces functional and proliferative abnormalities

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Notch activation differentially regulates renal progenitors proliferation and differentiation toward the podocyte lineage in glomerular disorders

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

Tracing the origin of glomerular extracapillary lesions from parietal epithelial cells.

21 January 2018

1 reference

https://api.crossref.org/works/10.1093%2FNDT%2FGFT413

21 January 2018

Identifiers

DOI

10.1093/NDT/GFT413

1 reference

11 May 2018

1 reference

11 May 2018