(Q53265500)

12 May 2018

title

Differential expression of alpha-synuclein isoforms in dementia with Lewy bodies. (English)

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12 May 2018

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1

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author name string

Lao JI

2

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Carrato C

3

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Mate JL

4

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López D

5

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Ferrer I

6

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Ariza A

7

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publication date

1 December 2004

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12 May 2018

Neuropathology and Applied Neurobiology

published in

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12 May 2018

volume

30

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page(s)

601-607

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issue

6

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Report of the second dementia with Lewy body international workshop: diagnosis and treatment. Consortium on Dementia with Lewy Bodies

cites work

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Mutation in the alpha-synuclein gene identified in families with Parkinson's disease

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Cellular co-localization of phosphorylated tau- and NACP/alpha-synuclein-epitopes in lewy bodies in sporadic Parkinson's disease and in dementia with Lewy bodies

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Dopaminergic loss and inclusion body formation in alpha-synuclein mice: implications for neurodegenerative disorders

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Expression of alpha-synuclein in the human brain: relation to Lewy body disease.

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Mice lacking alpha-synuclein display functional deficits in the nigrostriatal dopamine system

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The precursor protein of non-A beta component of Alzheimer's disease amyloid is a presynaptic protein of the central nervous system

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Microtubule-associated protein 1B is a component of cortical Lewy bodies and binds alpha-synuclein filaments

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Binding of alpha-synuclein to brain vesicles is abolished by familial Parkinson's disease mutation.

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NACP, a protein implicated in Alzheimer's disease and learning, is natively unfolded

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Membrane-bound alpha-synuclein has a high aggregation propensity and the ability to seed the aggregation of the cytosolic form

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Quantifying mRNA in postmortem human brain: influence of gender, age at death, postmortem interval, brain pH, agonal state and inter-lobe mRNA variance

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Altered expression of the synuclein family mRNA in Lewy body and Alzheimer's disease

21 January 2018

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21 January 2018

Identifiers

DOI

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12 May 2018

PubMed ID

15541000

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12 May 2018